Chapter 14. Pathogenesis of Cardiovascular Complications in the Acquired Immunodeficiency Syndrome

  1. Ronald Ross Watson1,2 and
  2. Douglas F. Larson3
  1. Giuseppe Barbaro

Published Online: 15 APR 2008

DOI: 10.1002/9780470692325.ch14

Immune Dysfunction and Immunotherapy in Heart Disease

Immune Dysfunction and Immunotherapy in Heart Disease

How to Cite

Barbaro, G. (2008) Pathogenesis of Cardiovascular Complications in the Acquired Immunodeficiency Syndrome, in Immune Dysfunction and Immunotherapy in Heart Disease (eds R. R. Watson and D. F. Larson), Blackwell Publishing Ltd, Oxford, UK. doi: 10.1002/9780470692325.ch14

Editor Information

  1. 1

    Department of Nutritional Sciences, The University of Arizona; and Sarver Heart Center, College of Medicine, The University of Arizona

  2. 2

    Division of Health Promotion Sciences, Mel and Enid Zuckerman Arizona College of Public Health, The University of Arizona Tucson, AZ USA

  3. 3

    Sarver Heart Center College of Medicine The University of Arizona Tucson, AZ USA

Publication History

  1. Published Online: 15 APR 2008
  2. Published Print: 30 AUG 2007

ISBN Information

Print ISBN: 9781405155687

Online ISBN: 9780470692325

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Keywords:

  • pathogenesis;
  • immunodeficiency syndrome;
  • cardiovascular system;
  • pathogenetic impact;
  • myocarditis

Summary

This chapter contains section titled:

  • Introduction

  • HIV-associated cardiomyopathy

  • Pathologic features

  • Myocarditis

  • Nonviral myocarditis

  • Viral myocarditis

  • Myocardial cytokine expression

  • Autoimmunity as a contributor to HIV-associated cardiomyopathy

  • Nutritional deficiencies as a factor in left ventricular dysfunction

  • Autonomic dysfunction as a factor in left ventricular dysfunction

  • Left ventricular dysfunction caused by drug cardiotoxicity

  • HIV infection, opportunistic infections, and vascular disease

  • HIV-associated pulmonary hypertension

  • Cardiac involvement in AIDS-associated neoplasms

  • HAART-associated lipodystrophy and metabolic syndrome

  • Molecular mechanisms

  • Mitochondrial dysfunction and lipodystrophy

  • Adipocytokines and lipodystrophy

  • HAART and cardiovascular disease

  • Conclusions

  • References