Conflict of interest: none.
Circadian abnormalities in mouse models of smith–magenis syndrome: Evidence for involvement of RAI1
Version of Record online: 23 MAY 2013
Copyright © 2013 Wiley Periodicals, Inc.
American Journal of Medical Genetics Part A
Volume 161, Issue 7, pages 1561–1568, July 2013
How to Cite
2013. Circadian abnormalities in mouse models of Smith–Magenis syndrome: Evidence for involvement of RAI1. Am J Med Genet Part A 161A:1561–1568., , .
Declaration of interest statement: J.R.L. is a consultant for Athena Diagnostics, has stock ownership in 23andMe and Ion Torrent Systems and is a coinventor on multiple United States and European patents for DNA diagnostics. The Department of Molecular and Human Genetics derives revenue from clinical testing by high-resolution human genome analysis.
- Issue online: 19 JUN 2013
- Version of Record online: 23 MAY 2013
- Manuscript Accepted: 22 FEB 2013
- Manuscript Received: 18 OCT 2012
- National Institute of Neurological Disorders and Stroke. Grant Number: R01 NS058529
- National Institutes of Health
- SMS Foundation
- Burroughs Wellcome Fund
- Alexander-von-Humboldt Foundation
Additional supporting information may be found in the online version of this article.
|ajmga35941-sm-0001-SuppFig-1.tif||170K||Supplemental Figure 1. Regressional analysis of wheel running activity and period length (tau/τ) in Dexras1+/- and Dexras1-/- mice and their wild-type littermates. No significant correlation between activity and tau values was observed for mutant or wild-type mice. R2 values are indicated on the graph adjacent to linear regression lines of best fit for Dexras1+/- (blue; n=17), wild-type (WT, green; n=14), and Dexras1-/- (red; n=21) mice.|
Please note: Wiley Blackwell is not responsible for the content or functionality of any supporting information supplied by the authors. Any queries (other than missing content) should be directed to the corresponding author for the article.