SEARCH

SEARCH BY CITATION

Cited in:

CrossRef

This article has been cited by:

  1. 1
    F.L. Mastaglia, A. Rojana-udomsart, I. James, M. Needham, T.J. Day, L. Kiers, J.A. Corbett, A.M. Saunders, M.W. Lutz, A.D. Roses, Polymorphism in the TOMM40 gene modifies the risk of developing sporadic inclusion body myositis and the age of onset of symptoms, Neuromuscular Disorders, 2013, 23, 12, 969

    CrossRef

  2. 2
    Philip B Verghese, Joseph M Castellano, David M Holtzman, Apolipoprotein E in Alzheimer's disease and other neurological disorders, The Lancet Neurology, 2011, 10, 3, 241

    CrossRef

  3. 3
    Brent T. Harris, Carrie A. Mohila, Essential Muscle Pathology for the Rheumatologist, Rheumatic Disease Clinics of North America, 2011, 37, 2, 289

    CrossRef

  4. 4
    Valerie Askanas, W. King Engel, Anna Nogalska, Inclusion Body Myositis: A Degenerative Muscle Disease Associated with Intra-Muscle Fiber Multi-Protein Aggregates, Proteasome Inhibition, Endoplasmic Reticulum Stress and Decreased Lysosomal Degradation, Brain Pathology, 2009, 19, 3
  5. 5
    Daniela L. Rebolledo, Alicia N. Minniti, Paula M. Grez, Ricardo Fadic, Rebecca Kohn, Nibaldo C. Inestrosa, Inclusion Body Myositis: A View from the Caenorhabditis elegans Muscle, Molecular Neurobiology, 2008, 38, 2, 178

    CrossRef

  6. 6
    M. Needham, F.L. Mastaglia, M.J. Garlepp, Genetics of inclusion-body myositis, Muscle & Nerve, 2007, 35, 5
  7. 7
    Michael R. Rose, Robert C. Griggs, Myopathies, 2007,

    CrossRef

  8. 8
    Aldobrando Broccolini, Teresa Gidaro, Roberta Morosetti, Carla Gliubizzi, Tiziana Servidei, Mario Pescatori, Pietro A. Tonali, Enzo Ricci, Massimiliano Mirabella, Neprilysin participates in skeletal muscle regeneration and is accumulated in abnormal muscle fibres of inclusion body myositis, Journal of Neurochemistry, 2006, 96, 3
  9. 9
    Valerie Askanas, W. King Engel, Molecular pathology and pathogenesis of inclusion-body myositis, Microscopy Research and Technique, 2005, 67, 3-4
  10. 10
    Gaetano Vattemi, W.King Engel, Janis McFerrin, Lucia Pastorino, Joseph D Buxbaum, Valerie Askanas, BACE1 and BACE2 in pathologic and normal human muscle, Experimental Neurology, 2003, 179, 2, 150

    CrossRef

  11. 11
    Ann M Reed, Steven R Ytterberg, Genetic and environmental risk factors for idiopathic inflammatory myopathies, Rheumatic Disease Clinics of North America, 2002, 28, 4, 891

    CrossRef

  12. 12
    Valerie Askanas, W. King Engel, Inclusion-body myositis and myopathies: different etiologies, possibly similar pathogenic mechanisms, Current Opinion in Neurology, 2002, 15, 5, 525

    CrossRef

  13. 13
    Zachary Simmons, Javad Towfighi, Sporadic Inclusion Body Myositis and Hereditary Inclusion Body Myopathy, Journal of Clinical Neuromuscular Disease, 2002, 3, 3, 122

    CrossRef

  14. 14
    Valerie Askanas, W. King Engel, Does Overexpression of βAPP in Aging Muscle Have a Pathogenic Role and a Relevance to Alzheimer's Disease?, The American Journal of Pathology, 1998, 153, 6, 1673

    CrossRef

  15. 15
    VALERIE ASKANAS, W. KING ENGEL, RENATE B. ALVAREZ, Fourteen Newly Recognized Proteins at the Human Neuromuscular Junctions-and Their Nonjunctional Accumulation in Inclusion-Body Myositisa, Annals of the New York Academy of Sciences, 1998, 841, 1
  16. 16
    Lee-Way Jin, Mark G. Hearn, Charles E. Ogburn, Ngocthao Dang, David Nochlin, Warren C. Ladiges, George M. Martin, Transgenic Mice Over-Expressing the C-99 Fragment of βPP with an α-Secretase Site Mutation Develop a Myopathy Similar to Human Inclusion Body Myositis, The American Journal of Pathology, 1998, 153, 6, 1679

    CrossRef

  17. 17
    Valerie Askanas, New developments in hereditary inclusion body myopathies, Annals of Neurology, 1997, 41, 4
  18. 18
    Michael J. Garlepp, Frank L. Mastaglia, Apolipoprotein E and inclusion body myositis, Annals of Neurology, 1996, 40, 6
  19. 19
    Valerie Askanas, W. King Engel, Anna Nogalska, Pathogenesis of Sporadic Inclusion-Body Myositis: Role of Aging and Muscle-Fiber Degeneration, and Accumulation of the Same Proteins as in Alzheimer and Parkinson Brains,