SEARCH

SEARCH BY CITATION

LITERATURE CITED

  • Cai X. 2006. Regulation of smooth muscle cells in development and vascular disease: current therapeutic strategies. Expert Rev Cardiovasc Ther 4: 789800.
  • Duckers HJ, Boehm M, True AL, Yet SF, San H, Park JL, Clinton Webb R, Lee ME, Nabel GJ, Nabel EG. 2001. Heme oxygenase-1 protects against vascular constriction and proliferation. Nat Med 7: 693698.
  • Durante W. 2011. Protective role of heme oxygenase-1 against inflammation in atherosclerosis. Front Biosci 17: 23722388.
  • Dzau VJ, Braun-Dullaeus RC, Sedding DG. 2002. Vascular proliferation and atherosclerosis: new perspectives and therapeutic strategies. Nat Med 8: 12491256.
  • Egozi D, Shapira M, Paor G, Ben-Izhak O, Skorecki K, Hershko DD. 2007. Regulation of the cell cycle inhibitor p27 and its ubiquitin ligase Skp2 in differentiation of human embryonic stem cells. FASEB J 21: 28072817.
  • Gordon D, Mohai LG, Schwartz SM. 1986. Induction of polyploidy in cultures of neonatal rat aortic smooth muscle cells. Circ Res 59: 633644.
  • Görlach A, Diebold I, Schini-Kerth VB, Berchner-Pfannschmidt U, Roth U, Brandes RP, Kietzmann T, Busse R. 2001. Thrombin activates the hypoxia-inducible factor-1 signaling pathway in vascular smooth muscle cells: Role of the p22 (phox)-containing NADPH oxidase. Circ Res 89: 4754.
  • Heldin CH, Westermark B. 1999. Mechanism of action and in vivo role of platelet-derived growth factor. Physiol Rev 79: 12831316.
  • Kedi X, Ming Y, Yongping W, Yi Y, Xiaoxiang Z. 2009. Free cholesterol overloading induced smooth muscle cells death and activated both ER- and mitochondrial-dependent death pathway. Atherosclerosis 207: 123130.
  • Kim JE, Kang YJ, Lee KY, Choi HC. 2009. Isoproterenol inhibits angiotensin II-stimulated proliferation and reactive oxygen species production in vascular smooth muscle cells through heme oxygenase-1. Biol Pharm Bull 32: 10471052.
  • Kreuzer J, Viedt C, Brandes RP, Seeger F, Rosenkranz AS, Sauer H, Babich A, Nürnberg B, Kather H, Krieger-Brauer HI. 2003. Platelet-derived growth factor activates production of reactive oxygen species by NAD(P)H oxidase in smooth muscle cells through Gi1,2. FASEB J 17: 3840.
  • Lassègue B, Sorescu D, Szöcs K, Yin Q, Akers M, Zhang Y, Grant SL, Lambeth JD, Griendling KK. 2001. Novel gp91 (phox) homologues in vascular smooth muscle cells: nox1 mediates angiotensin II-induced superoxide formation and redox-sensitive signaling pathways. Circ Res 88: 888894.
  • Liu XM, Peyton KJ, Ensenat D, Wang H, Schafer AI, Alam J, Durante W. 2005. Endoplasmic reticulum stress stimulates heme oxygenase-1 gene expression in vascular smooth muscle. Role in cell survival. J Biol Chem 280: 872877.
  • Majesky MW, Reidy MA, Bowen-Pope DF, Hart CE, Wilcox JN, Schwartz SM. 1990. PDGF ligand and receptor gene expression during repair of arterial injury. J Cell Biol 111: 21492158.
  • Martinet W, Croons V, Timmermans JP, Herman AG, De Meyer GR. 2007. Nitric oxide selectively depletes macrophages in atherosclerotic plaques via induction of endoplasmic reticulum stress. Br J Pharmacol 152: 493500.
  • Mori, K. 2000. Tripartite management of unfolded proteins in the endoplasmic reticulum. Cell 101: 451454.
  • Newby AC, Zaltsman AB. 2000. Molecular mechanisms in intimal hyperplasia. J Pathol 190: 300309.
  • Ohta S, Hattori Y, Nakanishi N, Sugimoto H, Kasai K. 2011. Differential modulation of immunostimulant-triggered NO production by endoplasmic reticulum stress inducers in vascular smooth muscle cells. J Cardiovasc Pharmacol 57: 434438.
  • Sprague AH, Khalil RA. 2009. Inflammatory cytokines in vascular dysfunction and vascular disease. Biochem Pharmacol 78: 539552.
  • Tenhunen R, Marver HS, Schmid R. 1968. The enzymatic conversion of heme to bilirubin by microsomal heme oxygenase. Proc Natl Acad Sci 61: 748755.
  • Tulis DA. 2006. Methods for identifying cardiovascular agents: a review. Recent Pat Cardiovasc Drug Discov 1: 4756.
  • Werstuck GH, Khan MI, Femia G, Kim AJ, Tedesco V, Trigatti B, Shi Y. 2006. Glucosamine-induced endoplasmic reticulum dysfunction is associated with accelerated atherosclerosis in a hyperglycemic mouse model. Diabetes 55: 93101.
  • Werstuck GH, Lentz SR, Dayal S, Hossain GS, Sood SK, Shi YY, Zhou J, Maeda N, Krisans SK, Malinow MR, Austin RC. 2001. Homocysteine-induced endoplasmic reticulum stress causes dysregulation of the cholesterol and triglyceride biosynthetic pathways. J Clin Invest 107: 12631273.
  • Xu C, Bailly-Maitre B, Reed JC. 2005. Endoplasmic reticulum stress: cell life and death decisions. J Clin Invest 115: 26562664.
  • Xue X, Piao JH, Nakajima A, Sakon-Komazawa S, Kojima Y, Mori K, Yagita H, Okumura K, Harding H, Nakano H. 2005. Tumor necrosis factor α (TNFα) induces the unfolded protein response (UPR) in a reactive oxygen species (ROS)-dependent fashion, and the UPR counteracts ROS accumulation by TNFα. J Biol Chem 280: 3391733925.
  • Yang X, Thomas DP, Zhang X, Culver BW, Alexander BM, Murdoch WJ, Rao MN, Tulis DA, Ren J, Sreejayan N. 2006. Curcumin inhibits platelet-derived growth factor-stimulated vascular smooth muscle cell function and injury-induced neointima formation. Arterioscler Thromb Vasc Biol 26: 8590.
  • Yumei Z, Shokei K, Yasukatsu I, Yasuhiro I, Takafumi N, Hitoshi M, Hiroshi I. 2003. Role of JNK, p38, and ERK in platelet-derived growth factor-induced vascular proliferation, migration, and gene expression. Arterioscler Thromb Vasc Biol 23: 795801.
  • Zhang K, Kaufman RJ. 2004. Signaling the unfolded protein response from the endoplasmic reticulum. J Biol Chem 279: 2593525938.
  • Zhang M, Zhang BH, Chen L, An W. 2002. Overexpression of heme oxygenase-1 protects smooth muscle cells against oxidative injury and inhibits cell proliferation. Cell Res 12: 123132.