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REFERENCES

  • 1
    Oppenheim JJ, Zachariae CO, Mukaida N, Matsushima K. Properties of the novel proinflammatory supergene “intercrine” cytokine family. Annu Rev Immunol 1991; 9: 61748.
  • 2
    Rollins BJ. Monocyte chemoattractant protein 1: a potential regulator of monocyte recruitment in inflammatory disease. Mol Med Today 1996; 2: 198204.
  • 3
    Volejnikova S, Laskari M, Marks SC Jr, Graves DT. Monocyte recruitment and expression of monocyte chemoattractant protein-1 are developmentally regulated in remodeling bone in the mouse. Am J Pathol 1997; 150: 171121.
  • 4
    Borzi RM, Mazzetti I, Macor S, Silvestri T, Bassi A, Cattini L, et al. Flow cytometric analysis of intracellular chemokines in chondrocytes in vivo: constitutive expression and enhancement in osteoarthritis and rheumatoid arthritis. FEBS Lett 1999; 455: 23842.
  • 5
    Cagnard N, Letourneur F, Essabbani A, Devauchelle V, Mistou S, Rapinat A, et al. Interleukin-32, CCL2, PF4F1 and GFD10 are the only cytokine/chemokine genes differentially expressed by in vitro cultured rheumatoid and osteoarthritis fibroblast-like synoviocytes. Eur Cytokine Netw 2005; 16: 28992.
  • 6
    Lisignoli G, Toneguzzi S, Pozzi C, Piacentini A, Riccio M, Ferruzzi A. Proinflammatory cytokines and chemokine production and expression by human osteoblasts isolated from patients with rheumatoid arthritis and osteoarthritis. J Rheumatol 1999; 26: 7919.
  • 7
    Langdon C, Leith J, Smith F, Richards CD. Oncostatin M stimulates monocyte chemoattractant protein-1– and interleukin-1–induced matrix metalloproteinase-1 production by human synovial fibroblasts in vitro. Arthritis Rheum 1997; 40: 213946.
  • 8
    De Hooge AS, van de Loo FA, Bennink MB, Arntz OJ, Fiselier TJ, Franssen MJ, et al. Growth plate damage, a feature of juvenile idiopathic arthritis, can be induced by adenoviral gene transfer of oncostatin M: a comparative study in gene-deficient mice. Arthritis Rheum 2003; 48: 175061.
  • 9
    Lin SK, Kok SH, Yeh FT, Kuo MY, Lin CC, Wang CC, et al. MEK/ERK and signal transducer and activator of transcription signaling pathways modulate oncostatin M–stimulated CCL2 expression in human osteoblasts through a common transcription factor. Arthritis Rheum 2004; 50: 78593.
  • 10
    Aaronson DS, Horvath CM. A road map for those who don't know JAK-STAT. Science 2002; 296: 16535.
  • 11
    Wen Z, Zhong Z, Darnell JE Jr. Maximal activation of transcription by Stat1 and Stat3 requires both tyrosine and serine phosphorylation. Cell 1995; 82: 24150.
  • 12
    Wagner BJ, Hayes TE, Hoban CJ, Cochran BH. The SIF binding element confers sis/PDGF inducibility onto the c-fos promoter. EMBO J 1990; 9: 447784.
  • 13
    Shyy JY, Lin MC, Han J, Lu Y, Petrime M, Chien S. The cis-acting phorbol ester “12-O-tetradecanoylphorbol 13-acetate”-responsive element is involved in shear stress-induced monocyte chemotactic protein 1 gene expression. Proc Natl Acad Sci U S A 1995; 92: 806973.
  • 14
    Valente AJ, Xie JF, Abramova MA, Wenzel UO, Abboud HE, Graves DT. A complex element regulates IFN-γ-stimulated monocyte chemoattractant protein-1 gene transcription. J Immunol 1998; 161: 371928.
  • 15
    Lee PJ, Camhi SL, Chin BY, Alam J, Choi AM. AP-1 and STAT mediate hyperoxia-induced gene transcription of heme oxygenase-1. Am J Physiol Lung Cell Mol Physiol 2000; 279: L17582.
  • 16
    Look DC, Pelletier MR, Tidwell RM, Roswit WT, Holtzman MJ. Stat1 depends on transcriptional synergy with Sp1. J Biol Chem 1995; 270: 302647.
  • 17
    Van der Pouw Kraan TC, van Gaalen FA, Kasperkovitz PV, Verbeet NL, Smeets TJ, Kraan MC, et al. Rheumatoid arthritis is a heterogeneous disease: evidence for differences in the activation of the STAT-1 pathway between rheumatoid tissues. Arthritis Rheum 2003; 48: 213245.
  • 18
    Huckel M, Schurigt U, Wagner AH, Stockigt R, Petrow PK, Thoss K, et al. Attenuation of murine antigen-induced arthritis by treatment with a decoy oligodeoxynucleotide inhibiting signal transducer and activator of transcription-1 (STAT-1). Arthritis Res Ther 2006; 8: R17.
  • 19
    Walker JG, Smith MD. The Jak-STAT pathway in rheumatoid arthritis. J Rheumatol 2005; 32: 16503.
  • 20
    Meydan N, Grunberger T, Dadi H, Shahar M, Arpaia E, Lapidot Z, et al. Inhibition of acute lymphoblastic leukaemia by a Jak-2 inhibitor. Nature 1996; 379: 6458.
  • 21
    Kumano K, Nakao A, Nakajima H, Miike S, Kurasawa K, Saito Y, et al. Blockade of JAK2 by tyrphostin AG-490 inhibits antigen-induced eosinophil recruitment into the mouse airways. Biochem Biophys Res Commun 2000; 270: 20914.
  • 22
    Dimitrova P, Ivanovska N. Tyrphostin AG-490 inhibited the acute phase of zymosan-induced inflammation. Int Immunopharmacol 2008; 8: 156777.
  • 23
    Stahl N, Boulton TG, Farruggella T, Ip NY, Davis S, Witthuhn BA, et al. Association and activation of Jak-Tyk kinases by CNTF-LIF-OSM-IL-6 β receptor components. Science 1994; 263: 925.
  • 24
    Nightingale J, Patel S, Suzuki N, Buxton R, Takagi KI, Suzuki J, et al. Oncostatin M, a cytokine released by activated mononuclear cells, induces epithelial cell-myofibroblast transdifferentiation via Jak/Stat pathway activation. J Am Soc Nephrol 2004; 15: 2132.
  • 25
    Tare RS, Oreffo RO, Clarke NM, Roach HI. Pleiotrophin/osteoblast-stimulating factor 1: dissecting its diverse functions in bone formation. J Bone Miner Res 2002; 17: 200920.
  • 26
    Zhou ZH, Chaturvedi P, Han YL, Aras S, Li YS, Kolattukudy PE, et al. IFN-γ induction of the human monocyte chemoattractant protein (hMCP)-1 gene in astrocytoma cells: functional interaction between an IFN-γ-activated site and a GC-rich element. J Immunol 1998; 160: 390816.
  • 27
    Cheng TH, Shih NL, Chen SY, Wang DL, Chen JJ. Reactive oxygen species modulate endothelin-I-induced c-fos gene expression in cardiomyocytes. Cardiovasc Res 1999; 41: 65462.
  • 28
    Lin SK, Chang HH, Chen YJ, Wang CC, Galson DL, Hong CY, et al. Epigallocatechin-3-gallate diminishes CCL2 expression in human osteoblastic cells via up-regulation of phosphatidylinositol 3-kinase/Akt/Raf-1 interaction: a potential therapeutic benefit for arthritis. Arthritis Rheum 2008; 58: 314556.
  • 29
    Xuan YT, Guo Y, Han Y, Zhu Y, Bolli R. An essential role of the JAK-STAT pathway in ischemic preconditioning. Proc Natl Acad Sci U S A 2001; 98: 90505.
  • 30
    Yamauchi K, Osuka K, Takayasu M, Usuda N, Nakazawa A, Nakahara N, et al. Activation of JAK/STAT signalling in neurons following spinal cord injury in mice. J Neurochem 2006; 96: 106070.
  • 31
    Wang Y, Kristan J, Hao L, Lenkoski CS, Shen Y, Matis LA. A role for complement in antibody-mediated inflammation: C5-deficient DBA/1 mice are resistant to collagen-induced arthritis. J Immunol 2000; 164: 43407.
  • 32
    Thwin MM, Douni E, Aidinis V, Kollias G, Kodama K, Sato K, et al. Effect of phospholipase A2 inhibitory peptide on inflammatory arthritis in a TNF transgenic mouse model: a time-course ultrastructural study. Arthritis Res Ther 2004; 6: R28294.
  • 33
    Zhan Y, Brown C, Maynard E, Anshelevich A, Ni W, Ho IC, et al. Ets-1 is a critical regulator of Ang II-mediated vascular inflammation and remodeling. J Clin Invest 2005; 115: 250816.
  • 34
    Mc Gee MM, Campiani G, Ramunno A, Nacci V, Lawler M, Williams DC. Activation of the c-Jun N-terminal kinase (JNK) signaling pathway is essential during PBOX-6-induced apoptosis in chronic myelogenous leukemia (CML) cells. J Biol Chem 2002; 277: 183839.
  • 35
    Decker T, Kovarik P. Serine phosphorylation of STATs. Oncogene 2000; 19: 262837.
  • 36
    Chung J, Uchida E, Grammer TC, Blenis J. STAT3 serine phosphorylation by ERK-dependent and -independent pathways negatively modulates its tyrosine phosphorylation. Mol Cell Biol 1997; 17: 650816.
  • 37
    Ala-aho R, Johansson N, Grenman R, Fusenig NE, Lopez-Otin C, Kahari VM. Inhibition of collagenase-3 (MMP-13) expression in transformed human keratinocytes by interferon-γ is associated with activation of extracellular signal-regulated kinase-1,2 and STAT1. Oncogene 2000; 19: 24857.
  • 38
    Hay N, Sonenberg N. Upstream and downstream of mTOR. Genes Dev 2004; 18: 192645.
  • 39
    Yokogami K, Wakisaka S, Avruch J, Reeves SA. Serine phosphorylation and maximal activation of STAT3 during CNTF signaling is mediated by the rapamycin target mTOR. Curr Biol 2000; 10: 4750.
  • 40
    Zhang X, Wrzeszczynska MH, Horvath CM, Darnell JE Jr. Interacting regions in Stat3 and c-Jun that participate in cooperative transcriptional activation. Mol Cell Biol 1999; 19: 713846.
  • 41
    Burley SK, Kamada K. Transcription factor complexes. Curr Opin Struct Biol 2002; 12: 22530.
  • 42
    Xu W, Comhair SA, Zheng S, Chu SC, Marks-Konczalik J, Moss J, et al. STAT-1 and c-Fos interaction in nitric oxide synthase-2 gene activation. Am J Physiol Lung Cell Mol Physiol 2003; 285: L13748.