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Additional Supporting Information may be found in the online version of this article.

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ccd24724-sup-0001-SuppFig1A.tif1435KSupporting Information Figure 1A. Examples of mechanisms producing strut malapposition are shown. Figure 2a shows an example of strut malapposition due to localized vessel enlargement or positive remodeling, in which the malapposed vessel wall diameter is larger than the diameter of the perpendicular wall, to which the stent is well apposed. Figure 2b shows an example of strut underexpansion with a diameter less than the predicted diameter based on the manufacturer's specifications (predicted 3.3 mm). Figure 2c shows stent undersizing wherein the malapposed vessel diameter (3.9 mm) is larger than the predicted stent diameter (3.3 mm). It should be noted that malapposition may be caused by more than one mechanism in any patient.
ccd24724-sup-0002-SuppFig1B.tif1435KSupporting Information Figure 1B. Examples of mechanisms producing strut malapposition are shown. Figure 2a shows an example of strut malapposition due to localized vessel enlargement or positive remodeling, in which the malapposed vessel wall diameter is larger than the diameter of the perpendicular wall, to which the stent is well apposed. Figure 2b shows an example of strut underexpansion with a diameter less than the predicted diameter based on the manufacturer's specifications (predicted 3.3 mm). Figure 2c shows stent undersizing wherein the malapposed vessel diameter (3.9 mm) is larger than the predicted stent diameter (3.3 mm). It should be noted that malapposition may be caused by more than one mechanism in any patient.
ccd24724-sup-0003-SuppFig1C.tif1432KSupporting Information Figure 1C. Examples of mechanisms producing strut malapposition are shown. Figure 2a shows an example of strut malapposition due to localized vessel enlargement or positive remodeling, in which the malapposed vessel wall diameter is larger than the diameter of the perpendicular wall, to which the stent is well apposed. Figure 2b shows an example of strut underexpansion with a diameter less than the predicted diameter based on the manufacturer's specifications (predicted 3.3 mm). Figure 2c shows stent undersizing wherein the malapposed vessel diameter (3.9 mm) is larger than the predicted stent diameter (3.3 mm). It should be noted that malapposition may be caused by more than one mechanism in any patient.
ccd24724-sup-0004-SuppFig2A.tif8815KSupporting Information Figure 2A. This figure illustrates the OCT findings in a patient after rapid inflation/deflation and prolonged inflation and the increase in minimal stent diameter (MSD) and area (MSA) and improvement in strut apposition. Figure 3a shows a mid-LAD lesion pre-PCI and the angiographic results after rapid inflation (Figure 3b) and prolonged inflation (Figure 3c). Please note the lack of residual angiographic stenosis after rapid inflation. MSD after rapid inflation was 2.79 mm and MSA was 6.20 mm2 (Figures 3d and 3e) and after prolonged inflation MSD was 3.15 mm and MSA was 7.80 mm2 (Figures 3f and 3g). Also shown is the maximal strut malapposition distance which decreased from 0.25 mm after rapid inflation/deflation to 0.19 mm after prolonged inflation.
ccd24724-sup-0005-SuppFig2C.tif8815KSupporting Information Figure 2C. This figure illustrates the OCT findings in a patient after rapid inflation/deflation and prolonged inflation and the increase in minimal stent diameter (MSD) and area (MSA) and improvement in strut apposition. Figure 3a shows a mid-LAD lesion pre-PCI and the angiographic results after rapid inflation (Figure 3b) and prolonged inflation (Figure 3c). Please note the lack of residual angiographic stenosis after rapid inflation. MSD after rapid inflation was 2.79 mm and MSA was 6.20 mm2 (Figures 3d and 3e) and after prolonged inflation MSD was 3.15 mm and MSA was 7.80 mm2 (Figures 3f and 3g). Also shown is the maximal strut malapposition distance which decreased from 0.25 mm after rapid inflation/deflation to 0.19 mm after prolonged inflation.
ccd24724-sup-0006-SuppFig2D.tif4245KSupporting Information Figure 2D. This figure illustrates the OCT findings in a patient after rapid inflation/deflation and prolonged inflation and the increase in minimal stent diameter (MSD) and area (MSA) and improvement in strut apposition. Figure 3a shows a mid-LAD lesion pre-PCI and the angiographic results after rapid inflation (Figure 3b) and prolonged inflation (Figure 3c). Please note the lack of residual angiographic stenosis after rapid inflation. MSD after rapid inflation was 2.79 mm and MSA was 6.20 mm2 (Figures 3d and 3e) and after prolonged inflation MSD was 3.15 mm and MSA was 7.80 mm2 (Figures 3f and 3g). Also shown is the maximal strut malapposition distance which decreased from 0.25 mm after rapid inflation/deflation to 0.19 mm after prolonged inflation.
ccd24724-sup-0007-SuppFig2E.tif4245KSupporting Information Figure 2E. This figure illustrates the OCT findings in a patient after rapid inflation/deflation and prolonged inflation and the increase in minimal stent diameter (MSD) and area (MSA) and improvement in strut apposition. Figure 3a shows a mid-LAD lesion pre-PCI and the angiographic results after rapid inflation (Figure 3b) and prolonged inflation (Figure 3c). Please note the lack of residual angiographic stenosis after rapid inflation. MSD after rapid inflation was 2.79 mm and MSA was 6.20 mm2 (Figures 3d and 3e) and after prolonged inflation MSD was 3.15 mm and MSA was 7.80 mm2 (Figures 3f and 3g). Also shown is the maximal strut malapposition distance which decreased from 0.25 mm after rapid inflation/deflation to 0.19 mm after prolonged inflation.
ccd24724-sup-0008-SuppFig2F.tif1433KSupporting Information Figure 2F. This figure illustrates the OCT findings in a patient after rapid inflation/deflation and prolonged inflation and the increase in minimal stent diameter (MSD) and area (MSA) and improvement in strut apposition. Figure 3a shows a mid-LAD lesion pre-PCI and the angiographic results after rapid inflation (Figure 3b) and prolonged inflation (Figure 3c). Please note the lack of residual angiographic stenosis after rapid inflation. MSD after rapid inflation was 2.79 mm and MSA was 6.20 mm2 (Figures 3d and 3e) and after prolonged inflation MSD was 3.15 mm and MSA was 7.80 mm2 (Figures 3f and 3g). Also shown is the maximal strut malapposition distance which decreased from 0.25 mm after rapid inflation/deflation to 0.19 mm after prolonged inflation.
ccd24724-sup-0009-SuppFig2G.tif1434KSupporting Information Figure 2G. This figure illustrates the OCT findings in a patient after rapid inflation/deflation and prolonged inflation and the increase in minimal stent diameter (MSD) and area (MSA) and improvement in strut apposition. Figure 3a shows a mid-LAD lesion pre-PCI and the angiographic results after rapid inflation (Figure 3b) and prolonged inflation (Figure 3c). Please note the lack of residual angiographic stenosis after rapid inflation. MSD after rapid inflation was 2.79 mm and MSA was 6.20 mm2 (Figures 3d and 3e) and after prolonged inflation MSD was 3.15 mm and MSA was 7.80 mm2 (Figures 3f and 3g). Also shown is the maximal strut malapposition distance which decreased from 0.25 mm after rapid inflation/deflation to 0.19 mm after prolonged inflation.

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