Vincenzo Amodio and Maria Florencia Tevy contributed equally to this work.
Special Issue Techniques
Transactivation in Drosophila of human enhancers by human transcription factors involved in congenital heart diseases†
Version of Record online: 11 OCT 2011
Copyright © 2011 Wiley Periodicals, Inc.
Special Issue: Special Issue on Drosophila
Volume 241, Issue 1, pages 190–199, January 2012
How to Cite
Amodio, V., Tevy, M. F., Traina, C., Ghosh, T. K. and Capovilla, M. (2012), Transactivation in Drosophila of human enhancers by human transcription factors involved in congenital heart diseases. Dev. Dyn., 241: 190–199. doi: 10.1002/dvdy.22763
Re-use of this article is permitted in accordance with the Terms and Conditions set out at http://wileyonlinelibrary.com/onlineopen#OnlineOpen_Terms
- Issue online: 15 DEC 2011
- Version of Record online: 11 OCT 2011
- Manuscript Accepted: 10 SEP 2011
- Fondazione Telethon. Grant Number: 3021
- Fondazione San Paolo
- Fondazione Cassa di Risparmio di Cento
- Unknown funding agency
Additional Supporting Information may be found in the online version of this article.
|DVDY_22763_sm_SuppFig1.tif||1494K||Supp. Fig. S1. Pattern of GFP expression driven by the 24B-GAL4 driver. Detection of GFP expression by in situ hybridization in embryos carrying the 24B-GAL4 driver and the UAS-GFP transgene. A, B: Lateral (A) and dorsal (B) view of a germ band–extended embryo. C, D: Lateral (C) and dorsal (D) view of a germ band–retracted embryo. GFP is expressed in the whole mesoderm.|
|DVDY22763SuppFigS2.tif||17974K||Supp. Fig. S2. The eye phenotype caused by GATA4, Nkx2.5, TBX5, or pnr overexpression is 100% penetrant. Low-magnification images of flies overexpressing GATA4 (A), Nkx2.5 (B), TBX5 (C), or pnr (D) through the GMR-GAL4 driver at 18°C. All flies show narrow eyes.|
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