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Hyperactive BMP signaling induced by ALK2R206H requires type II receptor function in a Drosophila model for classic fibrodysplasia ossificans progressiva

  1. Viet Q. Le,
  2. Kristi A. Wharton*

Article first published online: 15 DEC 2011

DOI: 10.1002/dvdy.23708

Issue

Developmental Dynamics

Developmental Dynamics

Special Issue: Special Issue on Drosophila

Volume 241, Issue 1, page i, January 2012

Additional Information

How to Cite

Le, V. Q. and Wharton, K. A. (2012), Hyperactive BMP signaling induced by ALK2R206H requires type II receptor function in a Drosophila model for classic fibrodysplasia ossificans progressiva. Dev. Dyn., 241: i. doi: 10.1002/dvdy.23708

Author Information

  1. Department of Molecular Biology, Cell Biology, and Biochemistry, Brown University, Providence, Rhode Island

*Department of Molecular Biology, Cell Biology, and Biochemistry, Brown University, Providence, RI 02912

Publication History

  1. Issue published online: 15 DEC 2011
  2. Article first published online: 15 DEC 2011
  3. Manuscript Accepted: 10 OCT 2011

Funded by

  • NIH. Grant Numbers: GM068118, T32GM007601
  • The Center for Research in FOP and Related Disorders. Grant Number: Cali Family Grant

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Keywords:

  • BMP signaling;
  • ALK2;
  • ACVR1;
  • FOP;
  • receptor kinase activation;
  • Sax;
  • Gbb;
  • Dpp;
  • Drosophila;
  • type I receptor;
  • type II receptor

Abstract

Overexpression of human GATA4 in the Drosophila eyes through the GMR-GAL4 driver. All flies show narrow eyes. The original photograph has been duplicated for esthetic purposes to create 6 panels. From Amodio et al., Developmental Dynamics 241:205-213, 2012.

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