Parts of this manuscript were presented at the Behavior Genetics Association conference in Amsterdam, June 2007.
Bivariate analysis of disordered eating characteristics in adolescence and young adulthood†
Article first published online: 18 OCT 2010
Copyright © 2010 Wiley Periodicals, Inc.
International Journal of Eating Disorders
Volume 43, Issue 8, pages 751–761, December 2010
How to Cite
Munn, M. A., Stallings, M. C., Hyun Rhee, S., Sobik, L. E., Corley, R. P., Rhea, S. A. and Hewitt, J. K. (2010), Bivariate analysis of disordered eating characteristics in adolescence and young adulthood. Int. J. Eat. Disord., 43: 751–761. doi: 10.1002/eat.20854
- Issue published online: 10 NOV 2010
- Article first published online: 18 OCT 2010
- Manuscript Accepted: 27 JUN 2010
- National Institute of Drug Abuse. Grant Numbers: DA011015, DA012485
- National Institute of Child Health and Human Development. Grant Number: HD010333
- National Institute of Mental Health. Grant Numbers: T32MH016990, F31MH084466
- National Institute of Child Health and Human Development. Grant Number: T32HD007289
- eating disorders;
- young adulthood;
- bivariate analysis;
- extended twin design
We examined the etiology of two disordered eating characteristics.
Participants included 1,470 female adolescent and young adult twins and their female nontwin siblings. Phenotypic factor analyses of a seven-item eating pathology screening tool yielded two factors: weight and shape concerns and behaviors (WSCB) and binge eating (BE). Univariate and bivariate extended twin analyses (including cotwins and nontwin siblings) were used to estimate the magnitude of genetic and environmental influences on these characteristics.
Analyses indicated that individual differences in WSCB and BE could be explained by additive genetic influences (a2 = 0.43 (95% CI: 0.33–0.52) and 0.49 (95% CI: 0.36–0.58), respectively), with the remaining variance due to nonshared environmental influences. The genetic correlation between WSCB and BE was estimated at 0.64; the nonshared environmental correlation was estimated at 0.27.
These results corroborate previous findings on genetic and environmental influences on disordered eating characteristics and suggest that findings can be extended to nontwin populations. © 2010 by Wiley Periodicals, Inc. Int J Eat Disord 2010; 43:751–761