Dr. Kaye has received salary support from the University of Pittsburgh and the University of California, San Diego; Research funding/support from the NIMH; Research funding for an investigator initiated treatment study from Astra-Zeneca and consulting fees from Lundbeck and Merck and the Eating Disorder Center of Denver. In addition, there are honoraria for presentations from academic institutions and meetings, and compensation for grant review activities from the National Institutes of Health. Dr. Bailer has received salary support from the Hilda & Preston Davis Foundation and the Price Foundation. Dr. Mathis has received royalty payments for licensed technology from GE Healthcare and Neuroptix as well as research grant support from Neuroptix. Dr. Mathis also served as a consultant for Janssen/Elan, Wyeth/Pfizer, and Novartis over the past three years. Dr. Frankle has received salary support from the University of Pittsburgh, research funding/support from the NIMH and NIDA as well as research funding and consulting fees from Sepracor, Inc. Dr. Duvvuri has received salary support from a Hilda & Preston Davis Foundation Fellowship in Eating Disorders Research and from a NIMH T32 fellowship. The remaining authors, Dr. Narendran and M. Himes declare that, except for income received from their primary employers and the aforementioned funding, no further financial support or compensation has been received from any individual or corporate entity over the past 3 years for research or professional service and there are no personal financial holdings that could be perceived as constituting a potential conflict of interest.
Version of Record online: 3 MAY 2011
Copyright © 2011 Wiley Periodicals, Inc.
International Journal of Eating Disorders
Volume 45, Issue 2, pages 263–271, March 2012
How to Cite
Bailer, U. F., Narendran, R., Frankle, W. G., Himes, M. L., Duvvuri, V., Mathis, C. A. and Kaye, W. H. (2012), Amphetamine induced dopamine release increases anxiety in individuals recovered from anorexia nervosa. Int. J. Eat. Disord., 45: 263–271. doi: 10.1002/eat.20937
Supported by MH046001, MH042984, K05-MH001894 from the National Institute of Mental Health and by the Prince Foundation.
- Issue online: 7 FEB 2012
- Version of Record online: 3 MAY 2011
- Manuscript Accepted: 13 MAR 2011
- 1American Psychiatric Association. Diagnostic and Statistical Manual of Mental Disorders, 4th ed. Text Revision (DSM-IV-TR). Washington, DC: American Psychiatric Association, 2000.
- 17Interaction between serotonin transporter and dopamine D2/D3 receptor radioligand measures is associated with harm avoidant symptoms in anorexia and bulimia nervosa (submitted)., , , , ,Mathis CA, , et al.
- 37Acute d-amphetamine challenge in schozophrenia: effects on cerebral glucose utilization and clinical symptomatology. Psychol Med 1994; 35: 649–657., , , , , , et al.
- 41Statistical Power Analysis for Behavioral Sciences. Hillsdale, NJ: Lawrence Erlbaum Associates, Inc, 1988..
- 53Family-genetic perspectives on anorexia nervosa and bulimia nervosa. In: Brownell K,Fairburn C, editors. Eating Disorders and Obesity-A Comprehensive Handbook. New York: The Guilford Press, 1995, pp. 212–218..