A novel feedback mechanism by Ephrin-B1/B2 in T-cell activation involves a concentration-dependent switch from costimulation to inhibition
Version of Record online: 23 MAY 2012
© 2012 WILEY-VCH Verlag GmbH & Co. KGaA, Weinheim
European Journal of Immunology
Volume 42, Issue 6, pages 1562–1572, June 2012
How to Cite
Kawano, H., Katayama, Y., Minagawa, K., Shimoyama, M., Henkemeyer, M. and Matsui, T. (2012), A novel feedback mechanism by Ephrin-B1/B2 in T-cell activation involves a concentration-dependent switch from costimulation to inhibition. Eur. J. Immunol., 42: 1562–1572. doi: 10.1002/eji.201142175
- Issue online: 8 JUN 2012
- Version of Record online: 23 MAY 2012
- Manuscript Accepted: 16 FEB 2012
- Manuscript Revised: 25 JAN 2012
- Manuscript Received: 13 OCT 2011
- Grants-in-Aid for the Scientific Research from the Ministry of Education, Culture, Sports
- Science and Technology in Japan (MEXT). Grant Number: #20012033
- Japan Society for the Promotion of Science (JSPS). Grant Number: #21591243
- Ministry of Health, Welfare
- Labor in Japan. Grant Number: H22-GANNRINSHO-Ippan032
Disclaimer: Supplementary materials have been peer-reviewed but not copyedited.
Figure 1. Fluorescence-activated cell sorter (FACS) analysis of spleen cells from RA/EG and RA/EG × CD11cCre mice.
Figure 2. Comparison of HIF1αflox, cHIF1αCCL17, and cHIF1αCD11c bone marrow derived dendritic cell (BMDC) for expression of maturation markers.
Please note: Wiley Blackwell is not responsible for the content or functionality of any supporting information supplied by the authors. Any queries (other than missing content) should be directed to the corresponding author for the article.