EBV stimulates TLR- and autophagy-dependent pathways and impairs maturation in plasmacytoid dendritic cells: Implications for viral immune escape
Article first published online: 30 OCT 2012
© 2012 WILEY-VCH Verlag GmbH & Co. KGaA, Weinheim
European Journal of Immunology
Volume 43, Issue 1, pages 147–158, January 2013
How to Cite
Severa, M., Giacomini, E., Gafa, V., Anastasiadou, E., Rizzo, F., Corazzari, M., Romagnoli, A., Trivedi, P., Fimia, G. M. and Coccia, E. M. (2013), EBV stimulates TLR- and autophagy-dependent pathways and impairs maturation in plasmacytoid dendritic cells: Implications for viral immune escape. Eur. J. Immunol., 43: 147–158. doi: 10.1002/eji.201242552
- Issue published online: 16 JAN 2013
- Article first published online: 30 OCT 2012
- Accepted manuscript online: 19 SEP 2012 07:08AM EST
- Manuscript Accepted: 12 SEP 2012
- Manuscript Revised: 7 AUG 2012
- Manuscript Received: 21 MAR 2012
- Italian Ministry of Health
- Ricerca Finalizzata 2007
- Italian Multiple Sclerosis Foundation. Grant Number: # 2009/R/7
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Figure S1. TLR7- and TLR9-dependent IFN-a production in pDCs treated by EBV.
Figure S2. Infection of primary B cells by EBV-GFP.
Figure S3. Enhancement of B7-H1 and ICOS-L expression in pDCs stimulated with a TLR9 agonist in the presence of EBV pDCs were treated for 48 h with CpG alone or in the presence of EBV.
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