These authors contributed equally to this work.
CRIg signals induce anti-intracellular bacterial phagosome activity in a chloride intracellular channel 3-dependent manner
Article first published online: 31 JAN 2013
© 2013 WILEY-VCH Verlag GmbH & Co. KGaA, Weinheim
European Journal of Immunology
Volume 43, Issue 3, pages 667–678, March 2013
How to Cite
Kim, K. H., Choi, B. K., Song, K. M., Cha, K. W., Kim, Y. H., Lee, H., Han, I.-S. and Kwon, B. S. (2013), CRIg signals induce anti-intracellular bacterial phagosome activity in a chloride intracellular channel 3-dependent manner. Eur. J. Immunol., 43: 667–678. doi: 10.1002/eji.201242997
- Issue published online: 11 MAR 2013
- Article first published online: 31 JAN 2013
- Accepted manuscript online: 26 DEC 2012 08:47AM EST
- Manuscript Accepted: 20 DEC 2012
- Manuscript Revised: 14 NOV 2012
- Manuscript Received: 18 SEP 2012
- National Cancer Center, Korea. Grant Number: NCC-1210370-1
- National Research Foundation of Korea (NRF). Grant Numbers: 2005-084-E00001, 2006-2004212
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Figure S1. CRIg signaling facilitates clearance of LM in monocytes.
Figure S2. Killing of intracellular LM by CRIg signaling is NO- and ROS-independent.
Figure S3. CLIC3 binds to the cytoplasmic domain of CRIg.
Figure S4. Generation of CLIC3−/− mice.
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