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Disclaimer: Supplementary materials have been peer-reviewed but not copyedited.

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gepi21745-sup-0001-Figs1.doc1144K

Figure S1. Genome-wide linkage results for baseline (black) and follow-up (grey) clinic visits for C. pneumoniae.

Figure S2. Genome-wide linkage results for baseline (black) and follow-up (grey) clinic visits for H. pylori.

Figure S3. Genome-wide linkage results for baseline (black) and follow-up (grey) clinic visits for CMV.

Figure S4. Genome-wide linkage results for baseline (black) and follow-up (grey) clinic visits for HSV-1.

Figure S5. Genome-wide linkage results for baseline (black) and follow-up (grey) clinic visits for HSV-2.

Figure S6. Genome-wide bivariate linkage results for C. pneumoniae for baseline and follow-up clinic visits.

Figure S7. Genome-wide bivariate linkage results for H. pylori for baseline and follow-up clinic visits.

Figure S8. Genome-wide bivariate linkage results for CMV for baseline and follow-up clinic visits.

Figure S9. Genome-wide bivariate linkage results for HSV-1 for baseline and follow-up clinic visits.

Figure S10. Genome-wide bivariate linkage results for HSV-2 for baseline and follow-up clinic visits.

gepi21745-sup-0002-table1.doc104KTable S1. Mean IgG antibody levels for pathogens examined in this study.
gepi21745-sup-0003-table2.doc53KTable S2. Bivariate linkage analysis of IgG antibody level traits at two time points: maximum LOD scores for suggestive (LOD ≥ 2.00) results.
gepi21745-sup-0004-table3.doc118KTable S3. Previous GWAS findings in regions with significant (LOD ≥ 2.86) and/or suggestive (LOD ≥ 2.00) results from multipoint linkage analysis of IgG antibody level traits.

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