Version of Record online: 10 AUG 2009
Copyright © 2009 American Association for the Study of Liver Diseases
Volume 50, Issue 6, pages 1773–1782, December 2009
How to Cite
Hösel, M., Quasdorff, M., Wiegmann, K., Webb, D., Zedler, U., Broxtermann, M., Tedjokusumo, R., Esser, K., Arzberger, S., Kirschning, C. J., Langenkamp, A., Falk, C., Büning, H., Rose-John, S. and Protzer, U. (2009), Not interferon, but interleukin-6 controls early gene expression in hepatitis B virus infection. Hepatology, 50: 1773–1782. doi: 10.1002/hep.23226
See Editorial on Page 1692.
Potential conflict of interest: Nothing to report.
- Issue online: 20 NOV 2009
- Version of Record online: 10 AUG 2009
- Accepted manuscript online: 10 AUG 2009 12:00AM EST
- Manuscript Accepted: 3 AUG 2009
- Manuscript Received: 2 APR 2009
- Deutsche Forschungsgemeinschaft. Grant Numbers: SFB 670 / SFB 576, SFB 415, RO632/13
- Helmholtz Alliance on Immunotherapy of Cancer. Grant Number: HA-202
- Cluster of Excellence “Inflammation at Interfaces
Additional Supporting Information may be found in the online version of this article.
|HEP_23226_sm_SupFig1.tif||529K||Supporting Fig. 1. Detection of HBV DNA replicative intermediates. (A) Southern blot analysis of HBV DNA intermediates in untreated HBV-infected cells, in rIL-6-treated cells (15 ng/ml) or after preincubation of rIL-6 with IL-6ab (200 ng/ml). rc - relaxed circular HBV DNA, ss –single stranded HBV DNA. (B) Quantification of HBV DNA intermediates by LightCycler PCR. HBV DNA levels were normalized to those for the reference PLAT gene and calculated by LightCyclerTM Relative Quantification software. HBV DNA levels determined in untreated cells were set to 100%. Values are shown as medium ± SD of three independent experiments.|
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