Version of Record online: 26 APR 2013
Copyright © 2013 American Association for the Study of Liver Diseases
Volume 57, Issue 6, pages 2287–2298, June 2013
How to Cite
Chen, J., Chan, A. W.H., To, K.-F., Chen, W., Zhang, Z., Ren, J., Song, C., Cheung, Y.-S., Lai, P. B.S., Cheng, S.-H., Ng, M. H.L., Huang, A. and Ko, B. C.B. (2013), SIRT2 overexpression in hepatocellular carcinoma mediates epithelial to mesenchymal transition by protein kinase B/glycogen synthase kinase-3β/β-catenin signaling. Hepatology, 57: 2287–2298. doi: 10.1002/hep.26278
Potential conflict of interest: Nothing to report.
This work is supported by the Research Grant Council (grant nos.: CUHK 466109 and 467210) and the National Natural Science Foundation of China (grant no.; 81272764) to B.C.B.K; and National Science and Technology Major Project of China (grant no.:2013ZX10002002, 2012ZX10002005) to A.H.
- Issue online: 12 JUN 2013
- Version of Record online: 26 APR 2013
- Accepted manuscript online: 24 JAN 2013 12:31PM EST
- Manuscript Accepted: 9 DEC 2012
- Manuscript Received: 21 AUG 2012
Additional Supporting Information may be found in the online version of this article.
|HEP_26278_sm_SuppFig1.tif||808K||Supporting Information Figure 1. Effect of SIRT2 gene silencing on the growth of HCC cells. Proliferation of HCC cells was suppressed by the depletion of SIRT2. PLC5, Huh-7 and HepG2 cells were transduced with the respective lentivirus and cell number was determined by trypan blue exclusion assay on day 6 (*, p<0.001).|
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