Correction

Errata

This article corrects:

  1. Receptor for activated C kinase 1 promotes hepatocellular carcinoma growth by enhancing mitogen-activated protein kinase kinase 7 activity Volume 57, Issue 1, 140–151, Article first published online: 7 January 2013

In the article titled “Receptor for Activated C Kinase 1 Promotes Hepatocellular Carcinoma Growth by Enhancing Mitogen-Activated Protein Kinase Kinase 7 Activity” (HEPATOLOGY 2013 Jan;57(1):140-151. doi: 10.1002/hep.25978) by Yuanyuan Guo, Wendie Wang, Jing Wang, Jiannan Feng, Qingyang Wang, Jianfeng Jin, Ming Lv, Xinying Li, Yan Li, Yuanfang Ma, Beifen Shen, and Jiyan Zhang, please note the following corrections:

The corresponding addresses are as follows: Jiyan Zhang, Ph.D., M.D., Institute of Basic Medical Sciences, E-mail: zhangjy@nic.bmi.ac.cn; Beifen Shen, Ph.D., Institute of Basic Medical Sciences, E-mail: shenbeifen@yahoo.com.cn; or Yuanfang Ma, Ph.D., Henan University, E-mail: Mayf@henu.edu.cn.

The correct figure legend for Figure 3 is as follows:

Fig. 3. RACK1 contributes to enhanced levels of P-MKK7/P-JNK in human HCC cells. (A-C) Forty-eight hours after transfection of HepG2 cells with RACK1 siRNA or control (Ctrl) nontargeting siRNA (A), or 96 hours after infection of HepG2, Huh7, or SK-Hep-1 cells with control lentivirus or lentivirus carrying RACK1 shRNA 1# (B), or HepG2 single clones stably expressing control shRNA or RACK1 shRNAs 2# and 3# were generated (C), cell lysates were then harvested and subjected to IB with the indicated Abs. (D and E) Forty-eight hours after transfection of HepG2 cells with mammalian expression vectors encoding GFP or GFP-RACK1 (D), or HepG2 single clones stably expressing FLAG-RACK1 and the mock control were generated (E), cell lysates were then harvested and subjected to IB with the indicated Abs.

In addition, the word “alternation”, which appears four times in the article, should be “alteration”.

The publisher regrets the error. DOI 10.1002/hep.26291

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