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Additional Supporting Information may be found in the online version of this article.

FilenameFormatSizeDescription
hep26644-sup-0001-suppfig1.tif1603KSupporting Information Figure 1: Comparison of clinical outcomes at 20 years, 25 years and 35 years after infection.
hep26644-sup-0002-suppfig2.tif1679KSupporting Information Figure 2: Evaluation of liver fibrosis at 35 years after infection by transient elastography. Transient elastography (FibroScan®, Echosens, Paris) was used for non-invasive fibrosis assessment. The measurements were performed according to the manufacturer's instructions (10 valid results required). Fibrosis stages were defined as METAVIR F2 with measurements >8.7 kPa, F3 (advanced fibrosis) >9.6 kPa and F4 (cirrhosis) >14.5 kPa.
hep26644-sup-0003-suppfig3.tif1595KSupporting Information Figure 3: Evaluation of liver fibrosis at 35 years after infection by liver biopsy. Liver biopsies were recommended to all viremic women to assess the individual inflammatory activity and fibrosis stage. Biopsies were scored for inflammation (grading 0-18) and fibrosis (staging 0-6) according to the Ishak score by the pathologists assigned to the respective centers.
hep26644-sup-0004-suppfig4.tif1878KSupporting Information Figure 4: Comparison of histological fibrosis stages at 20 years, 25 years and 35 years after infection. Biopsies were scored for inflammation (grading 0-18) and fibrosis (staging 0-6) according to the Ishak score by the pathologists assigned to the respective centers.
hep26644-sup-0005-suppfig5.tif1564KSupporting Information Figure 5: Interleukin 28B (IL28B) polymorphism in relation to the natural and treatment-induced course of HCV infection. The IL28B gene polymorphism rs12979860 was determined by real-time polymerase chain reaction and melting curve analysis.
hep26644-sup-0006-suppinfo.doc30KSupporting Information

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