Transmission of a homogenous hepatitis B virus population of A1896-containing strains leading to mild resolving acute hepatitis and seroconversion to hepatitis B e antigen antibodies in an adult
Article first published online: 30 DEC 2003
Copyright © 1997 by the American Association for the Study of Liver Diseases
Volume 26, Issue 3, pages 743–746, September 1997
How to Cite
Mphahlele, M. J., Shattock, A. G., Boner, W., Quinn, J., McCormick, P. A. and Carman, W. F. (1997), Transmission of a homogenous hepatitis B virus population of A1896-containing strains leading to mild resolving acute hepatitis and seroconversion to hepatitis B e antigen antibodies in an adult. Hepatology, 26: 743–746. doi: 10.1002/hep.510260329
- Issue published online: 30 DEC 2003
- Article first published online: 30 DEC 2003
- 1Mutation preventing formation of e antigen in patients with chronic HBV infection. Lancet 1989; ii: 588–591., , , , , , .
- 2Identification of HBV variants which cannot produce precore-derived HBeAg and may be responsible for severe hepatitis. Ital J Gastroenterol 1989; 21: 151–154., , , , , , .
- 9Fulminant hepatitis B: induction by hepatitis B virus mutants defective in the precore region and incapable of encoding e antigen. Gastroenterology 1991; 324: 1087–1094., , , , , , , et al.
- 18HBV core promotor mutations cause enhanced viral replication in association with fulminant hepatitis [Abstract]. Hepatology 1995; 22: 260A., , , .
- 20Distinct clinical outcomes and mutational patterns charaterize clusters of viruses from epidemiologically independent fulminant hepatitis B cases. 1997; submitted., , , , , .