Communicated by Richard Wooster
Version of Record online: 9 MAY 2008
This article is a US Government work, and, as such, is in the public domain in the United States of America. Published in 2008 by Wiley-Liss, Inc.
Volume 29, Issue 10, pages 1194–1208, October 2008
How to Cite
Boyle, J., Ueda, T., Oh, K.-S., Imoto, K., Tamura, D., Jagdeo, J., Khan, S. G., Nadem, C., DiGiovanna, J. J. and Kraemer, K. H. (2008), Persistence of repair proteins at unrepaired DNA damage distinguishes diseases with ERCC2 (XPD) mutations: cancer-prone xeroderma pigmentosum vs. non-cancer-prone trichothiodystrophy. Hum. Mutat., 29: 1194–1208. doi: 10.1002/humu.20768
This article is a US Government work, and, as such, is in the public domain in the United States of America.
- Issue online: 23 SEP 2008
- Version of Record online: 9 MAY 2008
- Manuscript Accepted: 28 JAN 2008
- Manuscript Received: 14 NOV 2007
- Intramural Research Program of the National Institutes of Health
- Center for Cancer Research, National Cancer Institute
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