Differential MicroRNA expression tracks neoplastic progression in inflammatory bowel disease-associated colorectal cancer
Article first published online: 13 APR 2012
© 2012 Wiley Periodicals, Inc.
Special Issue: Deep Phenotyping for Precision Medicine
Volume 33, Issue 5, page 899, May 2012
How to Cite
- Issue published online: 13 APR 2012
- Article first published online: 13 APR 2012
Vol. 33, Issue 3, 551–560, Article first published online: 14 FEB 2012
The original article to which this Erratum refers was published in Human Mutation 33(3):551–560 (DOI 10.1002/humu.22021).
The originally published version contained errors in the caption to Figure 1. The corrected caption is given here in full. The authors regret the errors.
Figure 1.A: Non-neoplastic Crohn disease (CD). Section showing chronically inflamed colonic mucosa without architectural change (magnification, 100×). The cytological features of dysplasia are absent. B: Crohn disease-associated (CD-a) dysplasia. Section of dysplasia in CD showing a tubulovillous adenoma-like dysplasia lesion (magnification, 100×). C: CD-a cancer. Invasive carcinoma in a CD patient showing infiltrating carcinoma glands set in tumor desmoplasia (magnification, 100×). D: Non-neoplastic ulcerative colitis (UC). Section showing chronically inflamed colonic mucosa with architectural change (magnification, 100×). The cytological features of dysplasia are absent. E: UC-associated (UC-a) dysplasia. High magnification view of part of a villous adenoma-like dysplastic lesion in a patient with UC (magnification, 200×). Note the full thickness stratification of nuclei with nuclei occupying the luminal portions on many of these colonic epithelial cells. F: UC-a colorectal cancer. Infiltrating moderately differentiated adenocarcinoma characterized by neoplastic glands showing an infiltration pattern set in tumor desmoplasia (magnification, 200×).