Supported by the Crohn's & Colitis Foundation of America through a generous gift from the Litwin Foundation, the Broad Medical Research Program, the Integrative Morphology core of the National Institutes of Health (NIH)-supported Cincinnati Children's Hospital Research Foundation Digestive Health Center (1P30DK078392-01), NIH grants R01 DK078683 (to LD.), DK068164 (to L.D.), and T32 DK007727 (to S.D., A.T.), and a VA Merit Review grant (to S.F.).
Innate dysfunction promotes linear growth failure in pediatric Crohn's disease and growth hormone resistance in murine ileitis†
Article first published online: 18 FEB 2011
Copyright © 2011 Crohn's & Crohn's & Colitis Foundation of America, Inc.
Inflammatory Bowel Diseases
Volume 18, Issue 2, pages 236–245, February 2012
How to Cite
D'Mello, S., Trauernicht, A., Ryan, A., Bonkowski, E., Willson, T., Trapnell, B. C., Frank, S. J., Kugasathan, S. and Denson, L. A. (2012), Innate dysfunction promotes linear growth failure in pediatric Crohn's disease and growth hormone resistance in murine ileitis. Inflamm Bowel Dis, 18: 236–245. doi: 10.1002/ibd.21689
- Issue published online: 10 JAN 2012
- Article first published online: 18 FEB 2011
- Manuscript Accepted: 19 JAN 2011
- Manuscript Received: 29 DEC 2010
- 10TNF promoter polymorphisms and modulation of growth retardation and disease severity in pediatric Crohn's disease. Am J Gastroenterol. 2005; 100: 1598–1604., , , et al.Direct Link:
- 13NOD2/CARD15 variants are associated with lower weight at diagnosis in children with Crohn's disease. Am J Gastroenterol. 2003; 98: 2479–2484., , , et al.Direct Link: