Funded by National Institutes of Health NIDDK R01 DK073992 (to E.M.Z.) and NICHD K12 HD028820 (to J.A.).
Computed tomography enterography findings correlate with tissue inflammation, not fibrosis in resected small bowel Crohn's disease†
Version of Record online: 24 JUN 2011
Copyright © 2011 Crohn's & Colitis Foundation of America, Inc.
Inflammatory Bowel Diseases
Volume 18, Issue 5, pages 849–856, May 2012
How to Cite
Adler, J., R. Punglia, D., Dillman, J. R., Polydorides, A. D., Dave, M., Al-Hawary, M. M., Platt, J. F., McKenna, B. J. and Zimmermann, E. M. (2012), Computed tomography enterography findings correlate with tissue inflammation, not fibrosis in resected small bowel Crohn's disease. Inflamm Bowel Dis, 18: 849–856. doi: 10.1002/ibd.21801
- Issue online: 12 APR 2012
- Version of Record online: 24 JUN 2011
- Manuscript Accepted: 20 MAY 2011
- Manuscript Received: 21 APR 2011
Additional Supporting Information may be found in the online version of this article.
|IBD_21801_sm_suppfig1.eps||710K||Supporting Figure 1 The three variables significantly associated with tissue fibrosis in the ordered logistic regression model were mucosal enhancement, mesenteric hypervascularity and inflammatory fat stranding. In this multi-panel graph, the y-axis of each panel represents the predicted probability of fibrosis for each ordinal level of tissue fibrosis from minimal fibrosis (blue) to transmural scar (red). As hypervascularity increases from the top panels to the lower panels, the predicted probability of minimal fibrosis increases. However increasing hypervascularity (along the x-axis) is associated with an increasing probability of more severe fibrosis. Increased inflammatory stranding is associated with a moderate increase in the predicted probability of more severe fibrosis.|
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