The first two authors contributed equally to this work.
Defective tumor necrosis factor release from Crohn's disease macrophages in response to toll-like receptor activation: Relationship to phenotype and genome-wide association susceptibility loci†
Version of Record online: 20 MAR 2012
Copyright © 2012 Crohn's & Colitis Foundation of America, Inc.
Inflammatory Bowel Diseases
Volume 18, Issue 11, pages 2120–2127, November 2012
How to Cite
Sewell, G. W., Rahman, F. Z., Levine, A. P., Jostins, L., Smith, P. J., Walker, A. P., Bloom, S. L., Segal, A. W. and Smith, A. M. (2012), Defective tumor necrosis factor release from Crohn's disease macrophages in response to toll-like receptor activation: Relationship to phenotype and genome-wide association susceptibility loci. Inflamm Bowel Dis, 18: 2120–2127. doi: 10.1002/ibd.22952
Re-use of this article is permitted in accordance with the Terms and Conditions set out at http://wileyonlinelibrary.com/onlineopen#OnlineOpen_Terms
- Issue online: 15 OCT 2012
- Version of Record online: 20 MAR 2012
- Manuscript Accepted: 17 FEB 2012
- Manuscript Received: 10 FEB 2012
- Wellcome Trust. Grant Number: (grant GHACB)
- National Association of Colitis and Crohn's disease
- 12Lipoteichoic acid (LTA) of Streptococcus pneumoniae and Staphylococcus aureus activates immune cells via Toll-like receptor (TLR)-2, lipopolysaccharide-binding protein (LBP), and CD14, whereas TLR-4 and MD-2 are not involved. J Biol Chem. 2003; 278: 15587–15594., , , et al.