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References

  • 1
    Koutroubakis I, Manousos ON, Meuwissen SG, et al. Environmental risk factors in inflammatory bowel disease. Hepatogastroenterology 1996; 43: 38193.
  • 2
    Elson CO, Cong Y, Brandwein S, et al. Experimental models to study molecular mechanisms underlying intestinal inflammation. Ann N Y Acad Sci 1998; 859: 8595.
  • 3
    Papadakis KA, Targan SR. Current theories on the causes of inflammatory bowel disease. Gastroenterol Clin North Am 1999; 28: 28396.
  • 4
    Hugot JP, Thomas G. Genome-wide scanning in inflammatory bowel diseases. Dig Dis Sci 1998; 16: 3649.
  • 5
    Hampe J, Schreiber S, Shaw SH, et al. A genome-wide analysis provides evidence for novel linkages in inflammatory bowel disease in a large European cohort. Am J Hum Genet 1999; 64: 80816.
  • 6
    Brandwein SL, McCabe RP, Cong Y, et al. Spontaneously colitic C3H/HeJBir mice demonstrate selective antibody reactivity to antigens of the enteric bacterial flora. J Immunol 1997; 159: 4452.
  • 7
    Cong Y, Brandwein SL, McCabe RP, et al. CD4+ T cells reactive to enteric bacterial antigens in spontaneously colitic C3H/HeJBir mice: increased T helper cell type 1 response and ability to transfer disease. J Exp Med 1998; 187: 85564.
  • 8
    Dianda L, Hanby AM, Wright NA, et al. T cell receptor-alpha beta-deficient mice fail to develop colitis in the absence of a microbial environment. Am J Pathol 1997; 150: 917.
  • 9
    Kullberg MC, Ward JM, Gorelick PL, et al. Helicobacter hepaticus triggers colitis in specific-pathogen-free interleukin-10 (IL-10)-deficient mice through an IL-12- and gamma interferon-dependent mechanism. Infect Immun 1998; 66: 515766.
  • 10
    Fox JG, Gorelick PL, Kullberg MC, et al. A novel urease-negative Helicobacter species associated with colitis and typhlitis in IL-10-deficient mice. Infect Immun 1999; 67: 175762.
  • 11
    Sellon RK, Tonkonogy S, Schultz M, et al. Resident enteric bacteria are necessary for development of spontaneous colitis and immune system activation in interleukin-10-deficient mice. Infect Immun 1998; 66: 522431.
  • 12
    Moore Kw, O'Garra A, de Waal Malefyt R, et al. Interleukin 10. Ann Rev Immunol 1993; 11: 16590.
  • 13
    Kühn R, Löhler J, Rennick D, et al. Interleukin-10-deficient mice develop chronic enterocolitis. Cell 1993; 75: 26374.
  • 14
    Lohler J, Kuhn R, Rennick D, et al. Interleukin-10-deficient mice: a model of chronic mucosal inflammation. In: TytgatG, BartelsmanJ, van DeventerS, eds. Inflammatory Bowel Disease. Vol. Falk Symposium 85. Boston: Kluwer Academic Publishers, 1995: 41018.
  • 15
    Rennick D, Davidson N, Berg D. Interleukin-10 gene knock-out mice: a model of chronic inflammation. Clin. Immunol. Immunopathol 1995; 76: S174178.
  • 16
    Berg DJ, Davidson N, Kühn R, et al. Enterocolitis and colon cancer in interleukin-10-deficient mice are associated with aberrant cytokine production and CD4+ TH1-like responses. J Clin Invest 1996; 98: 101020.
  • 17
    Sundberg JP, Elson CO, Bedigian HG, et al. Spontaneous, heritable colitis in a new substrain of C3H/HeJ mice. Gastroenterology 1994; 107: 172635.
  • 18
    Mähler M, Bristol IJ, Leiter EH, et al. Differential susceptibility of inbred mouse strains to dextran sulfate sodium-induced colitis. Am J Physiol 1998; 274: G544G551.
  • 19
    Mähler M, Bristol IJ, Sundberg JP, et al. Chromosomal location of genes determining susceptibility of mice to dextran sulfate sodium (DSS)-induced colitis. Genomics 1999; 55: 14756.
  • 20
    Green EL. Biology of the Laboratory Mouse. New York: Blakiston Division McGraw-Hill, 1966: 706.
  • 21
    Zheng XX, Steele AW, Nickerson PW, et al. Administration of noncytolytic IL-10/Fc in murine models of lipopolysaccharide-induced septic shock and allogeneic islet transplantation. J Immunol 1995; 154: 5590600.
  • 22
    Zheng XX, Steele AW, Hancock WW, et al. A noncytolytic IL-10/Fc fusion protein prevents diabetes, blocks autoimmunity, and promotes suppressor phenomena in NOD mice. J Immunol 1997; 158: 450713.
  • 23
    Elson C, Ealding W, Lefkowitz J. A lavage technique allowing repeated measurement of IgA antibody in mouse intestinal secretions. J Immunol Methods 1984; 67: 1018.
  • 24
    Powrie F, Leach M, Mauze S, et al. Phenotypically distinct subsets of CD4+ T cells induce or protect form chronic intestinal inflammation in C.B-17 scid mice. Int Immunol 1993; 5: 146171.
  • 25
    Horak I. Immunodeficiency in IL-2-knockout mice. Clin Immunol Immunpathol 1995; 76: S172S173.
  • 26
    Ma A, Datta M, Margosian E, et al. T cells, but not B cells, are required for bowel inflammation in interleukin 2-deficient mice. J Exp Med 1995; 182: 156772.
  • 27
    Sadlack B, Löhler J, Schorle H, et al. Generalized autoimmune disease in interleukin-2-deficient mice is triggered by an uncontrolled activation and proliferation of CD4+ T cells. Eur J Immunol 1995; 25: 30539.
  • 28
    Davidson NJ, Leach MW, Fort MM, et al. T helper cell 1-type CD4+ T cells, but not B cells, mediate colitis in interleukin-10-deficient mice. J Exp Med 1996; 184: 24151.
  • 29
    Hörnquist CE, Lu X, Rogers-Fani PM, et al. Gαi2-deficient mice with colitis exhibit a local increase in memory CD4+ T cells and pro-inflammatory Th1-type cytokines. J Immunol 1997; 158: 106877.
  • 30
    Takeda K, Clausen BE, Kaisho T, et al. Enhanced Th1 activity and development of chronic enterocolitis in mice devoid of Stat3 in macrophages and neutrophils. Immunity 1999; 10: 3949.
  • 31
    Duchmann R, Schmitt E, Knolle P, et al. Tolerance towards resident intestinal flora in mice is abrogated in experimental colitis and restored by treatment with interleukin-10 or antibodies to interleukin-12. Eur J Immunol 1996; 26: 93438.
  • 32
    Groux H, O'Garra A, Bigler M, et al. A CD4+ T-cell subset inhibits antigen-specific T-cell responses and prevents colitis. Nature 1997; 389: 73742.
  • 33
    Asseman C, Powrie F. Interleukin 10 is a growth factor for a population of regulatory T cells. Gut 1998; 42: 1578.
  • 34
    Dieleman L, Arends A, Tonkonogy S, et al. Interleukin-10 deficient mice monoassociated with Helicobacter hepaticus do not develop significant colitis. Gastroenterology 1999; 116: A700.
  • 35
    Cassatella MA. The neutrophil: one of the cellular targets of interleukin-10. Int J Clin Lab Res 1998; 28: 14861.
  • 36
    Cassatella MA. Neutrophil-derived proteins: selling cytokines by the pound [in process citation]. Adv Immunol 1999; 73: 369509.
  • 37
    Vetvicka V. C3H/HeJ mice. In: RihovaB, VetvickaV, eds. Immunological Disorders in Mice. Boca Raton: CRC Press Inc., 1991: 15371.