Cancer Cell Biology
Molecular mechanisms involved in activity of h7C10, a humanized monoclonal antibody, to IGF-1 receptor
Version of Record online: 26 NOV 2008
Copyright © 2008 Wiley-Liss, Inc.
International Journal of Cancer
Volume 124, Issue 10, pages 2281–2293, 15 May 2009
How to Cite
Broussas, M., Dupont, J., Gonzalez, A., Blaecke, A., Fournier, M., Corvaïa, N. and Goetsch, L. (2009), Molecular mechanisms involved in activity of h7C10, a humanized monoclonal antibody, to IGF-1 receptor. Int. J. Cancer, 124: 2281–2293. doi: 10.1002/ijc.24186
- Issue online: 16 MAR 2009
- Version of Record online: 26 NOV 2008
- Accepted manuscript online: 26 NOV 2008 12:00AM EST
- Manuscript Accepted: 11 NOV 2008
- Manuscript Received: 4 JUL 2008
Additional Supporting Information may be found in the online version of this article.
|IJC_24186_sm_suppfigure1.tif||824K||Supporting Figure 1. IGF-1R internalization and degradation upon h7C10 binding.|
|IJC_24186_sm_suppfigure2.tif||938K||Supporting Figure 2. Beta arrestin-1 is not involved in the h7C10-induced IGF-1R degradation.|
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