Cancer Genetics

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Tumor suppressor dual-specificity phosphatase 6 (DUSP6) impairs cell invasion and epithelial-mesenchymal transition (EMT)-associated phenotype

  1. Victor Chun Lam Wong1,†,
  2. Han Chen2,†,
  3. Josephine Mun Yee Ko1,
  4. Kwok Wah Chan3,
  5. Yuen Piu Chan3,
  6. Simon Law4,
  7. Daniel Chua1,
  8. Dora Lai-Wan Kwong1,
  9. Hong Lok Lung1,
  10. Gopesh Srivastava3,
  11. Johnny Cheuk On Tang5,
  12. Sai Wah Tsao6,
  13. Eugene R. Zabarovsky7,8,
  14. Eric J. Stanbridge9,
  15. Maria Li Lung1,‡,*

Article first published online: 20 APR 2011

DOI: 10.1002/ijc.25970

Issue

International Journal of Cancer

International Journal of Cancer

Volume 130, Issue 1, pages 83–95, 1 January 2012

Additional Information

How to Cite

Wong, V. C. L., Chen, H., Ko, J. M. Y., Chan, K. W., Chan, Y. P., Law, S., Chua, D., Kwong, D. L.-W., Lung, H. L., Srivastava, G., Tang, J. C. O., Tsao, S. W., Zabarovsky, E. R., Stanbridge, E. J. and Lung, M. L. (2012), Tumor suppressor dual-specificity phosphatase 6 (DUSP6) impairs cell invasion and epithelial-mesenchymal transition (EMT)-associated phenotype. Int. J. Cancer, 130: 83–95. doi: 10.1002/ijc.25970

Author Information

  1. 1

    Department of Clinical Oncology and Center for Cancer Research, University of Hong Kong, Hong Kong (SAR), People's Republic of China

  2. 2

    Division of Life Science, Hong Kong University of Science and Technology, Hong Kong (SAR), People's Republic of China

  3. 3

    Department of Pathology, University of Hong Kong, Hong Kong (SAR), People's Republic of China

  4. 4

    Department of Surgery, University of Hong Kong, Hong Kong (SAR), People's Republic of China

  5. 5

    Department of Applied Biology and Chemical Technology, Hong Kong Polytechnic University, Hong Kong SAR, People's Republic of China

  6. 6

    Department of Anatomy, University of Hong Kong, Hong Kong (SAR), People's Republic of China

  7. 7

    Department of Microbiology, Tumor and Cell Biology, Karolinska Institute, Stockholm, Sweden

  8. 8

    Laboratory of Structural and Functional Genomics, Engelhardt Institute of Molecular Biology, Russian Academy of Sciences, Moscow, 119991, Russia

  9. 9

    Department of Microbiology and Molecular Genetics, University of California, Irvine, CA

  1. †

    V.C.L.W. and H.C. contributed equally to this work.

  2. ‡

    Fax: 852-2819-5872,

*Room L2-23, 2/F, Laboratory Block, Department of Clinical Oncology, The University of Hong Kong, 21 Sassoon Road, Pokfulam, Hong Kong, HKSAR, People's Republic of China

  1. †

    V.C.L.W. and H.C. contributed equally to this work.

  2. ‡

    Fax: 852-2819-5872,

Publication History

  1. Issue published online: 27 OCT 2011
  2. Article first published online: 20 APR 2011
  3. Accepted manuscript online: 3 FEB 2011 01:59PM EST
  4. Manuscript Accepted: 30 DEC 2010
  5. Manuscript Received: 13 AUG 2010

Funded by

  • The University Grants Council Area of Excellence grant and the University of Hong Kong Seed Funding (MLL) Programme for Basic Research
  • the Swedish Cancer Society
  • the Swedish Research Council
  • the Swedish Institute
  • Karolinska Institute (ERZ)

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