The authors have declared that no conflict of interest exists.
L-cysteine and hydrogen sulfide increase PIP3 and AMPK/PPARγ expression and decrease ROS and vascular inflammation markers in high glucose treated human U937 monocytes
Article first published online: 15 AUG 2013
Copyright © 2013 Wiley Periodicals, Inc.
Journal of Cellular Biochemistry
Volume 114, Issue 10, pages 2334–2345, October 2013
How to Cite
Manna, P. and Jain, S. K. (2013), L-cysteine and hydrogen sulfide increase PIP3 and AMPK/PPARγ expression and decrease ROS and vascular inflammation markers in high glucose treated human U937 monocytes. J. Cell. Biochem., 114: 2334–2345. doi: 10.1002/jcb.24578
- Issue published online: 15 AUG 2013
- Article first published online: 15 AUG 2013
- Accepted manuscript online: 4 JUN 2013 06:16AM EST
- Manuscript Accepted: 18 APR 2013
- Manuscript Received: 15 MAR 2012
- Office of Dietary Supplements of the National Institutes of Health. Grant Number: RO1 DK072433
- Malcolm Feist Endowed Chair in Diabetes
- Malcolm Feist Cardiovascular Research Endowment
- LSU Health Sciences Center, Shreveport
Options for accessing this content:
- If you have access to this content through a society membership, please first log in to your society website.
- If you would like institutional access to this content, please recommend the title to your librarian.
- Login via other institutional login options http://onlinelibrary.wiley.com/login-options.
- You can purchase online access to this Article for a 24-hour period (price varies by title)
- If you already have a Wiley Online Library or Wiley InterScience user account: login above and proceed to purchase the article.
- New Users: Please register, then proceed to purchase the article.
Login via OpenAthens
Search for your institution's name below to login via Shibboleth.
Registered Users please login:
- Access your saved publications, articles and searches
- Manage your email alerts, orders and subscriptions
- Change your contact information, including your password
Please register to:
- Save publications, articles and searches
- Get email alerts
- Get all the benefits mentioned below!