Loss of Cbl–PI3K Interaction Enhances Osteoclast Survival due to p21-Ras Mediated PI3K Activation Independent of Cbl-b
Version of Record online: 7 MAY 2014
© 2014 Wiley Periodicals, Inc.
Journal of Cellular Biochemistry
Volume 115, Issue 7, pages 1277–1289, July 2014
How to Cite
Adapala, N. S., Barbe, M. F., Tsygankov, A. Y., Lorenzo, J. A. and Sanjay, A. (2014), Loss of Cbl–PI3K Interaction Enhances Osteoclast Survival due to p21-Ras Mediated PI3K Activation Independent of Cbl-b. J. Cell. Biochem., 115: 1277–1289. doi: 10.1002/jcb.24779
- Issue online: 7 MAY 2014
- Version of Record online: 7 MAY 2014
- Accepted manuscript online: 27 JAN 2014 07:45AM EST
- Manuscript Accepted: 24 JAN 2014
- Manuscript Received: 16 AUG 2013
- National Institute of health Grant. Grant Number: AR0550601
Additional supporting information may be found in the online version of this article at the publisher's web-site.
|jcb24779-sm-0001-SupFig-S1.tif||712K||Figure S1. Genotyping to verify the absence of Cbl-b and the presence of Y737F mutation in the YF/YF;Cbl-b−− mice.|
|jcb24779-sm-0002-SupFig-S2.tif||7957K||Figure S2. Increased numbers of osteoclasts in CYB mice with normal actin cytoskeleton.|
|jcb24779-sm-0003-SupFig-S3.tif||19880K||Figure S3. Two-dimensional images of tibial cortical region of 8-week-old male mice.|
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