This study was funded in part by a Pennsylvania Psychological Foundation Award.
The Role of Anxiety Sensitivity, Behavioral Inhibition, and Cognitive Biases in Anxiety Symptoms: Structural Equation Modeling of Direct and Indirect Pathways
Version of Record online: 6 JUL 2012
© 2012 Wiley Periodicals, Inc.
Journal of Clinical Psychology
Volume 68, Issue 10, pages 1122–1141, October 2012
How to Cite
Viana, A. G. and Gratz, K. L. (2012), The Role of Anxiety Sensitivity, Behavioral Inhibition, and Cognitive Biases in Anxiety Symptoms: Structural Equation Modeling of Direct and Indirect Pathways. J. Clin. Psychol., 68: 1122–1141. doi: 10.1002/jclp.21890
We thank students Anyaliese Hancock and Kali Falnes for their assistance.
- Issue online: 11 SEP 2012
- Version of Record online: 6 JUL 2012
- Pennsylvania Psychological Foundation Award
- anxiety sensitivity;
- behavioral inhibition;
- risk factors
Limited research has examined how temperamental (i.e., behavioral inhibition, anxiety sensitivity) and cognitive (i.e., interpretive and judgment biases) risks for the development anxiety covary to influence anxiety symptoms. Thus, the present study aimed to advance understanding of the direct and indirect links between anxiety sensitivity, behavioral inhibition, and interpretive biases, and judgment biases (in the form of perceived control) to anxiety outcomes (i.e., worry and trait anxiety symptoms).
842 emerging adults (mean = 18.75 years, standard deviation = 1.05; age range = 18–24; 70% women) recruited from a university in the northeast participated in this study. Participants completed a battery of self-report measures assessing risk factors and anxiety outcomes of interest.
Structural equation modeling revealed anxiety sensitivity and behavioral inhibition were directly linked with anxiety outcomes. Anxiety sensitivity and behavioral inhibition were also indirectly linked with anxiety outcomes through interpretive and judgment biases. The hypothesized model was partially invariant across high-risk and low-risk groups for anxiety disorders.
Results of this study provide preliminary support for theoretical models hypothesizing a developmental progression from temperamental to cognitive risks and culminating in the expression of anxiety symptoms. Limitations and clinical implications of this research are discussed. © 2012 Wiley Periodicals, Inc. J. Clin. Psychol. 00:1-20, 2012.