The MET receptor tyrosine kinase in invasion and metastasis
Article first published online: 2 JUL 2007
Copyright © 2007 Wiley-Liss, Inc.
Journal of Cellular Physiology
Volume 213, Issue 2, pages 316–325, November 2007
How to Cite
Benvenuti, S. and Comoglio, P. M. (2007), The MET receptor tyrosine kinase in invasion and metastasis. J. Cell. Physiol., 213: 316–325. doi: 10.1002/jcp.21183
- Issue published online: 27 AUG 2007
- Article first published online: 2 JUL 2007
- Manuscript Accepted: 21 MAY 2007
- Manuscript Received: 18 MAY 2007
Various cytokines and soluble growth factors upon interaction with their membrane receptors are responsible for inducing cellular proliferation, differentiation, movement, and protection from anoikis (a planned suicide activated by normal cells in absence of attachment to neighboring cells or extracellular matrix (EMC)). Among those soluble factors a major position is exerted by hepatocyte growth factor (HGF) together with its receptor MET and macrophage-stimulating protein (MSP) in cooperation with its receptor RON. J. Cell. Physiol. 213: 316–325, 2007. © 2007 Wiley-Liss, Inc.