This study was supported by a faculty research grant of Yonsei University College of Medicine for 2009 (6-2009-0200) to Sung Huhn Kim, MD. The authors have no other financial relationships, or conflicts of interest to disclose.
Gene regulation by glucocorticoid in ENaC-mediated Na+ transport by middle ear epithelial cells
Article first published online: 16 OCT 2013
Copyright © 2013 The American Laryngological, Rhinological and Otological Society, Inc.
Volume 124, Issue 2, pages E27–E33, February 2014
How to Cite
Kim, B. G., Kim, J. Y., Kim, M., Kim, C.-H., Choi, J. Y. and Kim, S. H. (2014), Gene regulation by glucocorticoid in ENaC-mediated Na+ transport by middle ear epithelial cells. The Laryngoscope, 124: E27–E33. doi: 10.1002/lary.24397
- Issue published online: 21 JAN 2014
- Article first published online: 16 OCT 2013
- Accepted manuscript online: 23 SEP 2013 05:11AM EST
- Manuscript Accepted: 19 AUG 2013
- Manuscript Revised: 2 AUG 2013
- Manuscript Received: 9 APR 2013
Additional Supporting Information may be found in the online version of this article.
|lary24397-sup-0001-suppInfo.tif||421K||Supplement 1. Transcript expression of 11β-HSD1 after dexamethasone treatment (100 nM for 12 h) in HMMECs (n = 6). Transcript expression of 11β-HSD1 was increased by 1.4 fold in the cells treated with dexamethasone for 12 hours when compared to the control, but this difference was not statistically significant. HMMECs = human middle ear epithelial cells.|
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