σN-dependent control of acid resistance and the locus of enterocyte effacement in enterohemorrhagic Escherichia coli is activated by acetyl phosphate in a manner requiring flagellar regulator FlhDC and the σS antagonist FliZ
Article first published online: 16 JUN 2014
© 2014 The Authors. MicrobiologyOpen published by John Wiley & Sons Ltd.
This is an open access article under the terms of the Creative Commons Attribution License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.
Volume 3, Issue 4, pages 497–512, August 2014
How to Cite
MicrobiologyOpen 2014; 3(4): 497–512
- Issue published online: 14 AUG 2014
- Article first published online: 16 JUN 2014
- Manuscript Accepted: 15 MAY 2014
- Manuscript Revised: 30 APR 2014
- Manuscript Received: 6 FEB 2014
|mbo3183-sup-0001-FigS1.tif||image/tif||772K||Figure S1. Effect of ntrB deletion on the expression of genes for GDAR and LEE control. Gene expression levels plotted for wild type (black), ΔrpoN (white), ΔntrC (hatched), and ΔntrB (gray). Asterisks denote significant difference between wild-type and respective mutants by t-test (*P < 0.05, **P < 0.01, n ≥ 3). Error bars denote standard deviation.|
|mbo3183-sup-0002-FigS2.tif||image/tif||364K||Figure S2. Growth curve for TW14359 and mutant derivative strains in DMEM. Optical density (OD600) is plotted for each strain as a function of time. Samples were taken every hour for 12 h. OD600 measurements differed by less than 5% for each time point and strain.|
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