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This article includes supplementary video clips, available online at http://www.interscience.wiley.com/jpages/0885-3185/suppmat.

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jws-mds.21384.mpg13628KSegment 1. Patient 2 displaying stridor during an episode of slow wave sleep. Right panel: polysomnographic recordings top to down traces = tracheal microphone; intercostalis muscle EMG surface activity; diaphragm EMG; oral nasal, thoracic and abdominal respirograms. Inset: O2 saturation.Segment 2. Patient 2. Awake fiberoptic laryngoscopy shows normal vocal cord abduction during inspiration.Segment 3. Patient 2. Fiberoptic laryngoscopy during sleep shows abnormal vocal cord adduction during inspiration with stridor. Right panel: polysomnographic recordings top to down traces = left thyroarytenoid (TA), right TA, left posterior cricoarytenoid (PCA), right PCA, tracheal microphone. Abnormal TA activation concomitant with physiologic PCA activity during inspiration.Segment 4. Patient 2 displaying stridor and Rem sleep behavior disorder during an episode of Rem sleep. Polysomnographic traces as per segment 1.Segment 5. Patient 1. Fiberoptic laryngoscopy shows normal vocal cord abduction during wakefulness.Segment 6. Patient 1. During sleep, fiberoptic laryngoscopy shows abnormal vocal cord adduction during inspiration with stridor. Left panel: polysomnographic traces from top to bottom = right and left thyroarytenoid (TA) and cricothyroid (CT) muscles, tracheal microphone. Abnormal diffuse activity associated with phasic activation of CT and TA muscles during inspiratory stridor.

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