This work was supported in part by the National Basic Science Program of China (81230028) and the National Science Foundation of China (81171183 and 2013CB966900), the Muscular Dystrophy Association, US National Institutes of Health (R01AI083294). All authors have nothing to report. S.F., L.Y., S. L., R.B., and F.D.S. recruited the patients, executed the treatment regimen, assessed the patients, and analyzed patients' data; S. F., Q.L., S.S., and N.S. processed the blood samples, performed flow cytometry, and analyzed the results; S.L., R.B., and F.D.S. formulated the study concept; S.L., Q.L., and R.B. edited the paper; F.D.S. provided funding and wrote the manuscript.
Interleukin-10 producing-B cells and their association with responsiveness to rituximab in myasthenia gravis
Article first published online: 27 FEB 2014
Published 2013 by Wiley Periodicals, Inc. This article is a US Government work and, as such, is in the public domain in the United States of America
Muscle & Nerve
Volume 49, Issue 4, pages 487–494, April 2014
How to Cite
Sun, F., Ladha, S. S., Yang, L., Liu, Q., Shi, S. X.-Y., Su, N., Bomprezzi, R. and Shi, F.-D. (2014), Interleukin-10 producing-B cells and their association with responsiveness to rituximab in myasthenia gravis. Muscle Nerve, 49: 487–494. doi: 10.1002/mus.23951
- Issue published online: 17 MAR 2014
- Article first published online: 27 FEB 2014
- Accepted manuscript online: 19 JUL 2013 05:13AM EST
- Manuscript Accepted: 4 JUL 2013
- Manuscript Revised: 3 JUL 2013
- Manuscript Received: 22 APR 2013
- 12In vivo production of interleukin-10 by non-T cells in rheumatoid arthritis, Sjogren's syndrome, and systemic lupus erythematosus. A potential mechanism of B lymphocyte hyperactivity and autoimmunity. Arthritis Rheum 1994;37:1647–1655., , , , , , et al.