This work was supported by grants for Health Science Research; the Center of Excellence (COE) program; the Human Frontier Science Program (HFSP); a Nervous and Mental Disorders grant from the Ministry of Health and Welfare, Japan; Japan Society for the Promotion of Science (JSPS); a grant-in-aid for JSPS fellows; the Ministry of Education, Culture, Sports, Science and Technology (MEXT), Japan; the Friends of Garrett Cumming Research Fund; the HM Toupin Neurological Science Research Fund; Parent Project Muscular Dystrophy (USA); Canada Foundation for Innovation; Alberta Advanced Education and Technology; University of Alberta; and Muscular Dystrophy Canada.
α1-Syntrophin–deficient mice exhibit impaired muscle force recovery after osmotic shock
Article first published online: 16 APR 2014
Copyright © 2013 Wiley Periodicals, Inc.
Muscle & Nerve
Volume 49, Issue 5, pages 728–735, May 2014
How to Cite
Yokota, T., Miyagoe-Suzuki, Y., Ikemoto, T., Matsuda, R. and Takeda, S. (2014), α1-Syntrophin–deficient mice exhibit impaired muscle force recovery after osmotic shock. Muscle Nerve, 49: 728–735. doi: 10.1002/mus.23990
- Issue published online: 16 APR 2014
- Article first published online: 16 APR 2014
- Accepted manuscript online: 20 AUG 2013 06:00AM EST
- Manuscript Accepted: 5 AUG 2013
- Manuscript Revised: 29 JUL 2013
- Manuscript Received: 9 DEC 2012
Additional Supporting Information may be found in the online version of this article.
|mus23990-sup-0001-suppinfo.tif||3254K||Fig S1. Lack of aquaporin-4 in α1-syntrophin deficient muscles The expression of α1-syntrophin (α1syn), aquaporin-4 (AQP4), and hematoxylin and eosin (H&E) staining in indicated mice.|
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