No conflicts of interest were declared.
Up-regulation of MKK4, MKK6 and MKK7 during prostate cancer progression: an important role for SAPK signalling in prostatic neoplasia†
Version of Record online: 19 JUN 2007
Copyright © 2007 Pathological Society of Great Britain and Ireland. Published by John Wiley & Sons, Ltd.
The Journal of Pathology
Volume 212, Issue 4, pages 386–394, August 2007
How to Cite
Lotan, T., Lyon, M., Huo, D., Taxy, J., Brendler, C., Foster, B., Stadler, W. and Rinker-Schaeffer, C. (2007), Up-regulation of MKK4, MKK6 and MKK7 during prostate cancer progression: an important role for SAPK signalling in prostatic neoplasia. J. Pathol., 212: 386–394. doi: 10.1002/path.2194
- Issue online: 5 JUL 2007
- Version of Record online: 19 JUN 2007
- Manuscript Accepted: 28 APR 2007
- Manuscript Revised: 20 APR 2007
- Manuscript Received: 13 MAR 2007
- University of Chicago Research Cure and Education Fund, USA. Grant Numbers: RO1 CA 89569, RO1 CA095367
- DOD Prostate Cancer Research Program, USAS. Grant Number: DAMD17-01-1-0700
Options for accessing this content:
- If you are a society or association member and require assistance with obtaining online access instructions please contact our Journal Customer Services team.
- If your institution does not currently subscribe to this content, please recommend the title to your librarian.
- Login via other institutional login options http://onlinelibrary.wiley.com/login-options.
- You can purchase online access to this Article for a 24-hour period (price varies by title)
- If you already have a Wiley Online Library or Wiley InterScience user account: login above and proceed to purchase the article.
- New Users: Please register, then proceed to purchase the article.
Login via OpenAthens
Search for your institution's name below to login via Shibboleth.
Registered Users please login:
- Access your saved publications, articles and searches
- Manage your email alerts, orders and subscriptions
- Change your contact information, including your password
Please register to:
- Save publications, articles and searches
- Get email alerts
- Get all the benefits mentioned below!