Additional Supporting Information may be found in the online version of this article.

pbc_24378_sm_SupplTab1.doc33KSupplementary Table S1
pbc_24378_sm_SupplTab2.docx107KSupplementary Table S2
pbc_24378_sm_SupplMetd.doc29KSupplementary Methods
pbc_24378_sm_SupplFigS1.tif7775KSupplementary Figure S1: Peer RCC was transfused on day 14 (36 ml/kg), 15 (10 ml/kg), 17 (10 ml/kg), and 21 (10 ml/kg). For the second intracranial hemorrhage on Day 72, RCC was transfused at 20 ml/kg. FFP was transfused on day 14 (10 ml/kg), 16 (10 ml/kg), and 17 (10 ml/kg) before the patient was diagnosed with FII deficiency. PCCs were administered as follows in FII units: Day 18 (40 U/kg), day 22 (80 U/kg), day 26 (80 U/kg), day 30 (100 U/kg), day 35 (50 U/kg), and day 40 (100 U/kg). On day 71 to 86, 100 U/kg was administered and after day 91, 130 U/kg was administered. We used head CTs to monitor his ICH. Head computed tomography was performed shown on the black arrow ([DOWNWARDS ARROW]).
pbc_24378_sm_SupplFigS2.tif11211KSupplementary Figure S2: Head computed tomography (CT) at the first intracranial hemorrhage in the left hemisphere (panel A). A widespread subdural hematoma was observed in the left temporal area and extended above and below the tentorium. A diffuse brain edema was observed and the density was entirely low. Head CT at the second intracranial hemorrhage in the right hemisphere (panel B). New subdural hematomas were observed in right temporal, occipital, and left parietal areas.

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