SEARCH

SEARCH BY CITATION

References

  • ALBRIGHT, T.D., JESSELL, T.M., KANDEL, E.R. & POSNER, M.I. (2000). Neural science: a century of progress and the mysteries that remain. Cell, 18 (Suppl. 100): 155.
  • BASLY, J., MARRE-FOURNIER, F., LE BAIL, J.C., HABRIOUX, G. & CHULIA, A.J. (2000). Estrogenic/Antiestrogenic and scavenging properties of (E)- and (Z)-resveratrol. Life Sci., 66, 769777.
  • CHASE, T.N. & OH, J.D. (2000). Striatal mechanisms and pathogenesis of parkinsonian signs and motor complications. Ann. Neurol., 47 (Suppl. 6738): 714.
  • CHOI, D.W. (1985). Glutamate neurotoxicity in cortical cell cultures is calcium dependent. Neurosci. Lett., 58, 293297.
  • CHOI, D.W. (1988). Glutamate neurotoxicity and disease of the nervous system. Neuron, 1, 623634.
  • CHOI, D.W., KOH, J.Y. & PETERS, S. (1988). Pharmacology of glutamate neurotoxicity in cortical cell culture: attenuation by NMDA antagonists. J. Neurosci., 8, 185196.
  • CHOI, D.W., MAULUCCI-GEDDE, M. & KRIEGSTEIN, A.R. (1987). Glutamate neurotoxicity in cortical cell culture. J. Neurosci., 7, 357368.
  • CHOI, D.W. & ROTHMAN, S.M. (1990). The role of glutamate neurotoxicity in hypoxic-ischemic neuronal death. Annu. Rev. Neurosci., 13, 171182.
  • CIABARRA, A.M., SULLIVAN, J.M., GAHN, L.G., PECHT, G., HEINEMANN, S. & SEVARINO, K.A. (1995). Cloning and characterization of chi-1: a developmentally regulated member of a novel class of the ionotropic glutamate receptor family. J. Neurosci., 15, 64986508.
  • CORSI, M., FINA, P. & TRIST, D.G. (1996). Co-agonism in drug-receptor interaction: illustrated by the NMDA receptors. Trends Pharmacol. Sci., 17, 220222.
  • COYLE, J.T. & PUTTFARCKEN, P. (1993). Oxidative stress, glutamate, and neurodegenerative disorders. Science, 262, 689695.
  • DANYSZ, W. & PARSONS, C.G. (1998). Glycine, NMDA receptors-physiological significance and possible therapeutic applications. Pharmacol. Rev., 50, 597664.
  • DAVIS, J.B. & MAHER, P. (1994). Protein kinase C activation inhibits glutamate-induced cytotoxicity in a neuronal cell line. Brain Res., 652, 169173.
  • DAWSON, V.L., BRAHMBHATT, H.P., MONG, J.A. & DAWSON, T.M. (1994). Expression of inducible nitric oxide synthase causes delayed neurotoxicity in primary mixed neuronal-glial cortical cultures. Neuropharmacology, 33, 14251430.
  • DOBLE, A. (1999). The role of excitotoxicity in neurodegenerative disease: Implications for therapy. Pharmacol. Ther., 81, 163221.
  • FLETCHER, E.J. & LODGE, D. (1988). Glycine reverses antagonism of N-methyl-D-aspartate by 1-hydroxy-3-aminopyrrolidone-2 (HA-966) but not by D-2-amino-5-phosphonovalerate (2-AP5) on rat cortical slices. Eur. J. Pharmacol., 151, 161162.
  • FOSTER, A.C. & KEMP, J.A. (1989). HA-966 antagonizes N-methyl-D-aspartate receptors through a selective interaction with the glycine modulatory site. J. Neurosci., 9, 21912196.
  • GILGUN-SHERKI, Y., MELAMED, E. & OFFERN, D. (2001). Oxidative stress induced-neurodegenerative disease: the need for antioxidants that penetrate the blood brain barrier. Neuropharmacology, 40, 59975.
  • GOODMAN, Y. & MATTSON, M.P. (1994). Selected forms of β-amyloid precursor protein protect hippocampal neurons against amyloid β-peptide induced oxidative injury. Exp. Neurol., 128, 112.
  • GRYNKIEWICZ, G., POENIE, M. & TSIEN, R.Y. (1985). A new generation of calcium indicators with greatly improved fluorescence properties. J. Biol. Chem., 260, 34403450.
  • HEINTZ, N. & ZOGHBI, H.Y. (2000). Insights from mouse models into the molecular basis of neurodegeneration. Annu. Rev. Physiol., 62, 779802.
  • HEWETT, S.J., ULIASZ, T.F., VIDWANS, A.S. & HEWETT, J.A. (2000). Cyclooxygenase-2 contributes to N-methyl-D-aspartate-mediated neuronal cell death in primary cortical cell culture. J. Pharm. Exp. Ther., 293, 417425.
  • KEMP, J.A., PRIESTLEY, T., LEESON, P.D., PRIESTLEY, T., TRIDGETT, R., IVERSEN, L.L. & WOODRUFF, G.N. (1988). 7-Chlorokynurenic acid is a selective antagonist at the glycine modulatory site of N-methyl-D-aspartate receptor complex. Proc. Natl. Acad. Sci. U.S.A., 85, 65476550.
  • KIM, S.R. & KIM, Y.C. (2000). Neuroprotective phenylpropanoid esters of rhamnose isolated from roots of Scrophularia buergeriana. Phytochemistry, 54, 503509.
  • KIM, Y.C., KIM, S.R., MARKELONIS, G.J. & OH, T.H. (1998). Ginsenosides Rb1 and Rg3 protect cultured rat cortical cells from glutamate-induced neurodegeneration. J. Neurosci. Res., 53, 426432.
  • KOH, J.Y. & CHOI, D.W. (1987). Quantitative determination of glutamate mediated cortical neuronal injury in cell culture by lactate dehydrogenase. J. Neurosci. Meth., 20, 8390.
  • LAFON-CAZAL, M., PIETRI, S., CULCASI, M. & BOCKAERT, J. (1993). NMDA-dependent superoxide production and neurotoxicity. Nature, 364, 535537.
  • LAUBE, B., HIRAI, H., STURGESS, M., BETZ, H. & KUHSE, J. (1997). Molecular determinants of agonist discrimination by NMDA receptor subunits: analysis of the glutamate binding site on the NR2B subunit. Neuron, 18, 493503.
  • LEE, J.M., ZIPFEL, G.J. & CHOI, D.W. (1999). The changing landscape of ischaemic brain injury mechanisms. Nature (London), 399 (Suppl. 6738): 714.
  • LIPTON, S.A. & ROSENBERG, P.A. (1994). Excitatory amino acids as a final common pathway for neurologic disorders. N. Eng. J. Med., 330, 613622.
  • LOWRY, O., ROSEBROUGH, H., FARR, A. & RANDALL, R. (1951). Protein measurement with Folin phenol reagent. J. Biol. Chem., 193, 265275.
  • MCDONALD, J.W. & JOHNSTON, M.V. (1990). Physiological and pathophysiological roles of excitatory amino acids during CNS development. Br. Res. Rev., 15, 4170.
  • MELDRUM, B. & GARTHWAITE, J. (1990). Excitatory amino acid neurotoxicity and neurodegenerative disease. Trends Pharmacol. Sci., 11, 379387.
  • MIYAMOTO, M., MURPHY, T.H., SCHNAAR, R.L. & COYLE, J.T. (1989). Antioxidants protect against glutamate-induced cytotoxicity in a neuronal cell line. J. Pharm. Exp. Ther., 250, 11321140.
  • MOLONEY, M.G. (1999). Excitatory amino acids. Nat. Prod. Rep., 16, 485498.
  • MUIR, K.W. & LEES, K.R. (1995). Clinical experience with excitatory amino acid antagonist drugs. Stroke, 26, 503513.
  • MURPHY, T.H., MIYAMOTO, M., SASTRE, A., SCHNAAR, R.L. & COYLE, J.T. (1989). Glutamate toxicity in a neuronal cell line involves inhibition of cystine transport leading to oxidative stress. Neuron, 2, 15471548.
  • NAKANISHI, N., SHNEIDER, N.A. & AXEL, R. (1990). A family of glutamate receptor genes: evidence for the formation of heteromultimeric receptors with distinct channel properties. Neuron, 5, 569581.
  • OLNEY, J.W. (1969). Brain lesions, obesity and other disturbances in mice treated with monosodium glutamate. Science, 164, 719721.
  • PREHN, J.H., LIPPERT, K. & KRIEGLSTEIN, J. (1995). Are NMDA or AMPA/kainate receptor antagonists more efficacious in the delayed treatment of excitotoxic neuronal injury? Eur. J. Pharmacol., 292, 179189.
  • QUARTAROLI, M., CARIGNANI, C., DAL FORNO, G., MUGNAINI, M., UGOLINI, A., ARBAN, R., BETTELINI, L., MARAIA, G., BELARDETTI, F., REGGIANI, A., TRIST, D.G., RATTI, E., DI FABIO, R. & CORSI, M. (1999). Potent antihyperalgesic activity without tolerance produced by glycine site antagonist of N-methyl-D-aspartate receptor GV19667A. J. Pharm. Exp. Ther., 290, 158169.
  • SHANGHAI, ACAD, SCI. & SHOGAKU, KAN., (eds) (1985). Dictionary of Chinese Herbal Medicine. Vol. 1–5, Japan, Shogukan Kan: Tokyo.
  • SINGH, L., DONALD, A.E., FOSTER, A.c. HUTSON, P.H., IVERSEN, L.L., IVERSON, D.S., KEMP, J.A., LEESON, P.D., MARSHALL, G.R., OLES, R.J., PRIESTLEY, T., THORN, L., TRICKLEBANK, M.D., VASS, C.A. & WILLIAMS, B.J. (1990). Enantiomers of HA-966 (3-amino-1-hydroxypyrrolid-2-one) exhibit distinct central nervous system effects: (+)-HA-966 is a selective glycine/N-methyl-D-aspartate receptor antagonist, but (−)-HA-966 is a potent γ-butyrolactone-like sedative. Proc. Natl. Acad. Sci. U.S.A., 87, 347351.
  • SUCHER, N.J., AWOBULUYI, M., CHOI, Y.B. & LIPTON, S.A. (1996). NMDA receptors: from genes to channels. Trends Pharmacol. Sci., 17, 348355.
  • TAMIZ, A.P., WHITTEMORE, E.R., ZHOU, Z.L., HUANG, J.C., DREWE, J.A., CHEN, J.C., CAI, S.X., WEBER, E., WOODWARD, R.M. & KEANA, J.F. (1998). Structure-activity relationships for a series of bis (phenylalkyl) amines: potent subtype-selective inhibitors of N-methyl-D-aspartate receptors. J. Med. Chem., 41, 34993506.
  • TRIST, D.G. (2000). Excitatory amino acid agonists and antagonists: pharmacology and therapeutic applications. Pharmaceutica Acta Helvetiae, 74, 221229.
  • WILLIAMS, K., CHAO, J., KASHIWAGI, K., MASUKO, T. & IGARASHI, K. (1996). Activation of N-methyl-D-aspartate receptors by glycine: role of an aspartate residue in the M3-M4 loop of the NR1 subunit. Mol. Pharmacol., 50, 701708.
  • WETTASINGHE, M. & SHAHIDI, F. (2000). Scavenging of reactive-oxygen species and DPPH free radicals by extracts of borage and evening primrose meals. Food Chem., 70, 1726.
  • ZHOU, L.M., GU, Z.Q., COSTA, A.M., YAMADA, K.A., MANSSON, P.E., GIORDANO, T., SKOLNICK, P. & JONES, K.A. (1997). (2S, 4R)-4-methylglutamic acid (SYM 2081): A selective, high affinity ligand for kainate receptors. J. Pharm. Exp. Ther., 280, 422427.