Disruption of Frontocerebellar Circuitry and Function in Alcoholism


  • Supported by the National Institute on Alcohol Abuse and Alcoholism [AA10723 (EVS); AA09272 (AJH); AA05592 (RP); AA10433 and AA10583 (PRM); AA05965 (AP)] and the National Institute of Mental Health [MH60234 (JED)], Bethesda, Maryland.

Edith V. Sullivan, PhD, Department of Psychiatry & Behavioral Sciences (5723), Stanford University School of Medicine, 401 Quarry Road, Stanford, CA 94305-5723; Fax: 650-859-2743; E-mail: edie@stanford.edu.


This article represents a symposium of the 2002 joint meeting of RSA and ISBRA held in San Francisco. Presentations were Neuropathology of alcohol-related cerebellar damage in humans, by Antony J. Harding; Neuropathological evidence of cerebellar damage in an animal model of alcoholism, by Roberta Pentney and Cynthia Dlugos; Understanding cortical-cerebellar circuits through neuroimaging study of chronic alcoholics, by Peter R. Martin and Mitchell H. Parks; and Functional reorganization of the brain in alcoholism: neuroimaging evidence, by John E. Desmond, S.H. Annabel Chen, Michelle R. Pryor, Eve De Rosa, Adolf Pfefferbaum, and Edith V. Sullivan.