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Epidermolysis bullosa pruriginosa triggered by scabies infestation

Authors

  • Jaehwan Kim,

    1. Department of Dermatology, St George Hospital, Sydney, New South Wales, Australia
    2. Faculty of Medicine, University of New South Wales, Sydney, New South Wales, Australia
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  • Chee Hoou Loh,

    1. Department of Dermatology, St George Hospital, Sydney, New South Wales, Australia
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  • Dedee F. Murrell

    Corresponding author
    1. Faculty of Medicine, University of New South Wales, Sydney, New South Wales, Australia
    • Department of Dermatology, St George Hospital, Sydney, New South Wales, Australia
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  • Conflict of interest: The authors declare that they have no conflicts of interest.

Correspondence: Dedee F. Murrell, M.A., B.M.B.Ch., F.A.A.D., M.D., F.A.C.D., Department of Dermatology, St George Hospital, Gray Street, Kogarah, Sydney, NSW 2217, Australia. Email: d.murrell@unsw.edu.au

Abstract

Epidermolysis bullosa pruriginosa (EB-Pr) is an unusual variant of dystrophic EB. Potential genetic disease modifiers and metabolic factors have been investigated, but thus far no specific insight into this phenotype has emerged. We report an in-depth description of three patients diagnosed as having EB-Pr in whom this particular phenotype developed after scabies infestation and dramatically improved after full treatment. This short communication suggests that scabies infestation could be one of the important triggering factors for the development of the EB-Pr phenotype.

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