β-amyloid context intensifies vascular smooth muscle cells induced inflammatory response and de-differentiation
Version of Record online: 1 APR 2013
© 2013 John Wiley & Sons Ltd and the Anatomical Society
Volume 12, Issue 3, pages 358–369, June 2013
How to Cite
Vromman, A., Trabelsi, N., Rouxel, C., Béréziat, G., Limon, I. and Blaise, R. (2013), β-amyloid context intensifies vascular smooth muscle cells induced inflammatory response and de-differentiation. Aging Cell, 12: 358–369. doi: 10.1111/acel.12056
- Issue online: 13 MAY 2013
- Version of Record online: 1 APR 2013
- Accepted manuscript online: 20 FEB 2013 02:10PM EST
- Manuscript Accepted: 4 FEB 2013
- Pierre Fabre Innovation
Table S1 Reagents and sources.
Fig. S1 Aβ1-40 effect on tumor necrosis factor-α (TNFα) -treated vascular smooth muscle cells (VSMCs).
Fig. S2 Expression of SM-Calponin and SM-MHC in vascular smooth muscle cells (VSMCs) treated by Aβ1-40 and IL1β.
Fig. S3 Conformational state of Aβ1-40 and its dose-effect on IL1β-induced vascular smooth muscle cells (VSMCs) inflammatory response.
Fig. S4 Aβ1-40 effect on the expression of the IL-1-R1 in vascular smooth muscle cells (VSMCs).
Fig. S5 Inflammatory molecules in vehicle- vs. Aβ1-40-treated endothelial cells culture medium.
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