Those authors contributed equally
High levels of oncomiR-21 contribute to the senescence-induced growth arrest in normal human cells and its knock-down increases the replicative lifespan
Article first published online: 19 APR 2013
© 2013 John Wiley & Sons Ltd and the Anatomical Society
Volume 12, Issue 3, pages 446–458, June 2013
How to Cite
Dellago, H., Preschitz-Kammerhofer, B., Terlecki-Zaniewicz, L., Schreiner, C., Fortschegger, K., Chang, M. W.-F., Hackl, M., Monteforte, R., Kühnel, H., Schosserer, M., Gruber, F., Tschachler, E., Scheideler, M., Grillari-Voglauer, R., Grillari, J. and Wieser, M. (2013), High levels of oncomiR-21 contribute to the senescence-induced growth arrest in normal human cells and its knock-down increases the replicative lifespan. Aging Cell, 12: 446–458. doi: 10.1111/acel.12069
- Issue published online: 13 MAY 2013
- Article first published online: 19 APR 2013
- Accepted manuscript online: 15 MAR 2013 09:15AM EST
- Manuscript Accepted: 21 FEB 2013
- Austrian Science Fund. Grant Number: P 24498-B20
- Genome Research Autria GEN-AU. Grant Number: 820982
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