Mitochondrial hormesis links low-dose arsenite exposure to lifespan extension
Article first published online: 6 MAY 2013
© 2013 John Wiley & Sons Ltd and the Anatomical Society
Volume 12, Issue 3, pages 508–517, June 2013
How to Cite
Schmeisser, S., Schmeisser, K., Weimer, S., Groth, M., Priebe, S., Fazius, E., Kuhlow, D., Pick, D., Einax, J. W., Guthke, R., Platzer, M., Zarse, K. and Ristow, M. (2013), Mitochondrial hormesis links low-dose arsenite exposure to lifespan extension. Aging Cell, 12: 508–517. doi: 10.1111/acel.12076
- Issue published online: 13 MAY 2013
- Article first published online: 6 MAY 2013
- Accepted manuscript online: 27 MAR 2013 10:40AM EST
- Manuscript Accepted: 16 MAR 2013
Fig. S1 Individual results for N2 lifespan and paraquat stress resistance assays.
Fig. S2 While arsenite accumulates within worms, low-dose arsenite exposure does not change food uptake and does not induce aversion behavior.
Fig. S3 Like N2 nematodes exposed to low-dose arsenite, mev-1 mutants display increased ROS formation but to a considerably larger extent.
Fig. S4 In silico promoter analyses of differentially expressed genes.
Table S1 Differentially expressed RNAs after 48 h of exposure to arsenite.
Table S2 Functional classification of upregulated DEGs (FunCat).
Table S3 Functional classification of upregulated DEGs (GO Term: Biological Process).
Data S1 Supporting experimental procedures.
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