Functional characterization of human CTC1 mutations reveals novel mechanisms responsible for the pathogenesis of the telomere disease Coats plus
Article first published online: 4 SEP 2013
© 2013 the Anatomical Society and John Wiley & Sons Ltd
Volume 12, Issue 6, pages 1100–1109, December 2013
How to Cite
Gu, P. and Chang, S. (2013), Functional characterization of human CTC1 mutations reveals novel mechanisms responsible for the pathogenesis of the telomere disease Coats plus. Aging Cell, 12: 1100–1109. doi: 10.1111/acel.12139
- Issue published online: 21 NOV 2013
- Article first published online: 4 SEP 2013
- Accepted manuscript online: 22 JUL 2013 04:18AM EST
- Manuscript Accepted: 10 JUL 2013
- NIA. Grant Number: R21 AG043747
Fig. S1 RT-PCR analysis of Flag-tagged WT and mutant CTC1 expression (top) and endogenous STN1 expression (bottom) in reconstituted CTC1−/− mouse embryonic fibroblasts.
Fig. S2 Stabilization of CTC1, STN1 and TEN1 requires CST complex formation.
Please note: Wiley Blackwell is not responsible for the content or functionality of any supporting information supplied by the authors. Any queries (other than missing content) should be directed to the corresponding author for the article.