Essential role for the TRF2 telomere protein in adult skin homeostasis
Version of Record online: 14 APR 2014
© 2014 The Authors. Aging Cell published by the Anatomical Society and John Wiley & Sons Ltd.
This is an open access article under the terms of the Creative Commons Attribution License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.
Volume 13, Issue 4, pages 656–668, August 2014
How to Cite
Martínez, P., Ferrara-Romeo, I., Flores, J. M. and Blasco, M. A. (2014), Essential role for the TRF2 telomere protein in adult skin homeostasis. Aging Cell, 13: 656–668. doi: 10.1111/acel.12221
- Issue online: 29 JUL 2014
- Version of Record online: 14 APR 2014
- Manuscript Accepted: 23 FEB 2014
- European Research Council (ERC) Project TEL STEM CELL. Grant Number: GA#232854
- European Union FP7 Projects (MARK-AGE) and (EuroBATS). Grant Numbers: 2007-A-20088, 2010-259749
- Spanish Ministry of Economy and Competitiveness Projects. Grant Numbers: SAF2008-05384, CSD2007-00017
- Regional of Government of Madrid Project (ReCaRe). Grant Number: S2010/BMD-2303
- AXA Research Fund (Life Risks Project)
- Lilly 2010 Preclinical Biomedicine Research Award (Fundación Lilly, Spain)
- Fundación Botín (Spain)
Fig. S1 Aberrant expression pattern of differentiation markers in TRF2-null epidermis.
Fig. S2 TRF2 deficiency leads to severe epidermal stem cell defects.
Fig. S3 53BP1 and p53 deficiencies do not rescue TRF2-associated proliferative defects.
Fig. S4 TRF2 deficiency does not impact in telomere length homeostasis in stratified epithelia.
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