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Data S1. Method and supplementary references.

Figure S1. Evolutionary conservation of theVDR gene. Analysis of the VDR gene including5′-2000 bp flanking transcriptional start and 3′-1000bp following final exon 9 were compared between human and mouse,rat and zebrafish.

Figure S2. VDR single nucleotidepolymorphism function. CD14+ monocytes of healthy individualshomozygous for the VDR GCT or AAC haplotype (BsmI,ApaI, TaqI order; GCT is frequent in the severeatopic dermatitis group, AAC in controls) were analysed exvivo or following calcitriol stimulation regarding VDR gene expression determined by quantitative polymerase chain reaction.

Table S1. Patient characteristics

Table S2. Primer sequences for VDR genotyping

Table S3. Product sizes of VDR single nucleotide polymorphisms by restriction fragment length polymorphism

Table S4. Haplotype of donors recruited for functional analysis

Table S5. Oligonucleotides used forquantitative reverse transcription–polymerase chainreaction

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