Bcl-3 deficiency protects against dextran-sodium sulphate-induced colitis in the mouse
Article first published online: 4 JUL 2013
© 2013 British Society for Immunology
Clinical & Experimental Immunology
Volume 173, Issue 2, pages 332–342, August 2013
How to Cite
O'Carroll, C., Moloney, G., Hurley, G., Melgar, S., Brint, E., Nally, K., Nibbs, R. J., Shanahan, F. and Carmody, R. J. (2013), Bcl-3 deficiency protects against dextran-sodium sulphate-induced colitis in the mouse. Clinical & Experimental Immunology, 173: 332–342. doi: 10.1111/cei.12119
- Issue published online: 4 JUL 2013
- Article first published online: 4 JUL 2013
- Accepted manuscript online: 15 APR 2013 05:31AM EST
- Manuscript Accepted: 9 APR 2013
- Science Foundation Ireland. Grant Numbers: 08/IN.1/B1843, 07/CE/B1368
- Marie Curie International Re-integration Grant programme
Fig. S1. Bcl-3 mRNA levels in normal (N, n = 11), Crohn's disease (CD, n = 10) and ulcerative colitis (UC, n = 10) colon tissue. Data extracted from the NCBI GEO data set GDS1330.
Fig. S2. Relative levels of cleaved caspase-3 normalized to β-actin levels in colon tissue from untreated (open bars) and dextran-sodium sulphate (DSS)-treated (filled bars) wild-type and Bcl-3−/−. Levels were quantified form immunoblot analysis presented in Fig. 6b in the main text.
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