Natural killer (NK) cell deficit in coronary artery disease: no aberrations in phenotype but sustained reduction of NK cells is associated with low-grade inflammation
Version of Record online: 3 DEC 2013
© 2013 British Society for Immunology
Clinical & Experimental Immunology
Volume 175, Issue 1, pages 104–112, January 2014
How to Cite
Backteman, K., Ernerudh, J. and Jonasson, L. (2014), Natural killer (NK) cell deficit in coronary artery disease: no aberrations in phenotype but sustained reduction of NK cells is associated with low-grade inflammation. Clinical & Experimental Immunology, 175: 104–112. doi: 10.1111/cei.12210
- Issue online: 3 DEC 2013
- Version of Record online: 3 DEC 2013
- Accepted manuscript online: 24 SEP 2013 06:43AM EST
- Manuscript Accepted: 16 SEP 2013
- Medical Research Council of Southeast Sweden
- Swedish Research Council
- Heart-Lung Foundation
- County of Östergötland
Figure S1. Fluorescence minus one (FMO) gating strategy.
Figure S2. Proportions of T cells, natural killer (NK) cells and subsets in patients and controls.
Table S1. Monoclonal antibodies and in-vivo lymphocyte profiles.
Table S2. Whole blood stimulation and detection of intracellular cytokines.
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