gtc12032-sup-0001-FigS1.pdfapplication/PDF327KFigure S1. Disappearance of the glycinergic mPSCs by GlyR blockade is likely not a result of remaining strychnine after washout.
gtc12032-sup-0002-FigS2.pdfapplication/PDF5437KFigure S2. Strychnine treatment with various concentrations also impaired GlyR clustering.
gtc12032-sup-0003-FigS3.pdfapplication/PDF4547KFigure S3. Glycinergic nerve terminals project on M-cell in larvae exposed to strychnine.
gtc12032-sup-0004-FigS4.pdfapplication/PDF1586KFigure S4. Expression of mCherry in M-cells in transgenic larvae
gtc12032-sup-0005-TblS1.pdfapplication/PDF301KTable S1. The GlyR clusters numbers and densities, and M-cell surface area in larvae shown in Fig. 2B, C, D
gtc12032-sup-0006-TblS2.pdfapplication/PDF299KTable S2. The densities of GlyR clusters following strychnine removal shown in Fig. 3B
gtc12032-sup-0007-TblS3.pdfapplication/PDF298KTable S3. The densities of GlyR clusters after 24 h-strychnine treatment shown in Fig. 4B
gtc12032-sup-0008-TblS4.pdfapplication/PDF300KTable S4. The densities of GlyR clusters after 1 h-strychnine treatment and washout of the strychnine shown in Fig. 5B
gtc12032-sup-0009-TblS5.pdfapplication/PDF301KTable S5. The densities of GlyR clusters after blockade of L-type Ca2+ channel or CaMKII shown in Fig. 6B, E
gtc12032-sup-0010-Supporting-Legends.docWord document53K 

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